Title : Chronic CSE treatment induces the growth of normal oral keratinocytes via PDK2 upregulation, increased glycolysis and HIF1α stabilization.

Pub. Date : 2011 Jan 19

PMID : 21283817






1 Functional Relationships(s)
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1 Furthermore, chronic CSE treatment was found to increase ROS (reactive oxygen species) production, and application of the ROS scavengers N-acetylcysteine abrogated the expression of PDK2 and HIF1alpha. Reactive Oxygen Species pyruvate dehydrogenase kinase 2 Homo sapiens