Title : Hepcidin as a therapeutic tool to limit iron overload and improve anemia in β-thalassemic mice.

Pub. Date : 2010 Dec

PMID : 21099112






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1 Here we demonstrate that a moderate increase in expression of hepcidin in beta-thalassemic mice limits iron overload, decreases formation of insoluble membrane-bound globins and reactive oxygen species, and improves anemia. reactive oxygen hepcidin antimicrobial peptide Mus musculus