Title : Anti-Inflammatory benefits of antibiotic-induced neutrophil apoptosis: tulathromycin induces caspase-3-dependent neutrophil programmed cell death and inhibits NF-kappaB signaling and CXCL8 transcription.

Pub. Date : 2011 Jan

PMID : 20956586






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1 In addition, tulathromycin significantly reduced levels of phosphorylated IkappaBalpha, nuclear translocation of NF-kappaB p65, and mRNA levels of proinflammatory interleukin-8 in lipopolysaccharide (LPS)-stimulated bovine neutrophils. tulathromycin NFKB inhibitor alpha Bos taurus