Title : Protective role of interleukin-17 in murine NKT cell-driven acute experimental hepatitis.

Pub. Date : 2010 Nov

PMID : 20847291






4 Functional Relationships(s)
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1 Here we show that during alpha-galactosylceramide (alphaGalCer)-induced hepatitis in mice, a model of hepatitis driven by specific activation of the innate immune system via NKT cells within the liver, NK1.1+ and CD4+ iNKT cells rapidly produce IL-17 and are the main IL-17-producing cells within the liver. alpha-galactosylceramide interleukin 17A Homo sapiens
2 Here we show that during alpha-galactosylceramide (alphaGalCer)-induced hepatitis in mice, a model of hepatitis driven by specific activation of the innate immune system via NKT cells within the liver, NK1.1+ and CD4+ iNKT cells rapidly produce IL-17 and are the main IL-17-producing cells within the liver. alpha-galactosylceramide interleukin 17A Homo sapiens
3 Here we show that during alpha-galactosylceramide (alphaGalCer)-induced hepatitis in mice, a model of hepatitis driven by specific activation of the innate immune system via NKT cells within the liver, NK1.1+ and CD4+ iNKT cells rapidly produce IL-17 and are the main IL-17-producing cells within the liver. alpha-galactosylceramide interleukin 17A Homo sapiens
4 Here we show that during alpha-galactosylceramide (alphaGalCer)-induced hepatitis in mice, a model of hepatitis driven by specific activation of the innate immune system via NKT cells within the liver, NK1.1+ and CD4+ iNKT cells rapidly produce IL-17 and are the main IL-17-producing cells within the liver. alpha-galactosylceramide interleukin 17A Homo sapiens