Pub. Date : 2010 Nov
PMID : 20847291
4 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
1 | Here we show that during alpha-galactosylceramide (alphaGalCer)-induced hepatitis in mice, a model of hepatitis driven by specific activation of the innate immune system via NKT cells within the liver, NK1.1+ and CD4+ iNKT cells rapidly produce IL-17 and are the main IL-17-producing cells within the liver. | alpha-galactosylceramide | interleukin 17A | Homo sapiens |
2 | Here we show that during alpha-galactosylceramide (alphaGalCer)-induced hepatitis in mice, a model of hepatitis driven by specific activation of the innate immune system via NKT cells within the liver, NK1.1+ and CD4+ iNKT cells rapidly produce IL-17 and are the main IL-17-producing cells within the liver. | alpha-galactosylceramide | interleukin 17A | Homo sapiens |
3 | Here we show that during alpha-galactosylceramide (alphaGalCer)-induced hepatitis in mice, a model of hepatitis driven by specific activation of the innate immune system via NKT cells within the liver, NK1.1+ and CD4+ iNKT cells rapidly produce IL-17 and are the main IL-17-producing cells within the liver. | alpha-galactosylceramide | interleukin 17A | Homo sapiens |
4 | Here we show that during alpha-galactosylceramide (alphaGalCer)-induced hepatitis in mice, a model of hepatitis driven by specific activation of the innate immune system via NKT cells within the liver, NK1.1+ and CD4+ iNKT cells rapidly produce IL-17 and are the main IL-17-producing cells within the liver. | alpha-galactosylceramide | interleukin 17A | Homo sapiens |