Title : Prominent role of KCa3.1 in endothelium-derived hyperpolarizing factor-type dilations and conducted responses in the microcirculation in vivo.

Pub. Date : 2009 Jun 1

PMID : 19218287






1 Functional Relationships(s)
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1 ACh-induced dilations in KCa3.1-/- mice were virtually abolished at low and intermediate concentrations and a remaining dilation at 10 micromol/L ACh was abrogated by blockade of KCa2.3 with UCL1684. Acetylcholine potassium intermediate/small conductance calcium-activated channel, subfamily N, member 4 Mus musculus