Title : Targeting MEK/MAPK signal transduction module potentiates ATO-induced apoptosis in multiple myeloma cells through multiple signaling pathways.

Pub. Date : 2008 Sep 15

PMID : 18583568






3 Functional Relationships(s)
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Protein Name
Organism
1 In HMCLs retaining a functional p53, PD treatment greatly enhances the ATO-induced p53 accumulation and p73, a p53 paralog, cooperates with p53 in caspase activation and apoptosis induction. Arsenic Trioxide tumor protein p53 Homo sapiens
2 In HMCLs retaining a functional p53, PD treatment greatly enhances the ATO-induced p53 accumulation and p73, a p53 paralog, cooperates with p53 in caspase activation and apoptosis induction. Arsenic Trioxide tumor protein p53 Homo sapiens
3 In HMCLs retaining a functional p53, PD treatment greatly enhances the ATO-induced p53 accumulation and p73, a p53 paralog, cooperates with p53 in caspase activation and apoptosis induction. Arsenic Trioxide tumor protein p53 Homo sapiens