Title : Gene-linked shift in ganglioside distribution influences growth and vascularity in a mouse astrocytoma.

Pub. Date : 2006 Sep

PMID : 16911584






3 Functional Relationships(s)
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1 Ganglioside distribution was altered in CT-2A tumor cells using an antisense construct to beta-1,4-N-acetylgalactosaminyltransferase (GalNAc-T), a key enzyme that uses the simple ganglioside GM3 as a substrate for the synthesis of the more complex gangliosides, GM2, GM1 and GD1a. Gangliosides beta-1,4-N-acetyl-galactosaminyltransferase 1 Homo sapiens
2 Ganglioside distribution was altered in CT-2A tumor cells using an antisense construct to beta-1,4-N-acetylgalactosaminyltransferase (GalNAc-T), a key enzyme that uses the simple ganglioside GM3 as a substrate for the synthesis of the more complex gangliosides, GM2, GM1 and GD1a. Gangliosides beta-1,4-N-acetyl-galactosaminyltransferase 1 Homo sapiens
3 Ganglioside distribution was altered in CT-2A tumor cells using an antisense construct to beta-1,4-N-acetylgalactosaminyltransferase (GalNAc-T), a key enzyme that uses the simple ganglioside GM3 as a substrate for the synthesis of the more complex gangliosides, GM2, GM1 and GD1a. Gangliosides beta-1,4-N-acetyl-galactosaminyltransferase 1 Homo sapiens