Title : PCBs exert an estrogenic effect through repression of the Wnt7a signaling pathway in the female reproductive tract.

Pub. Date : 2006 Jun

PMID : 16759992






4 Functional Relationships(s)
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1 Perinatal exposure to high concentrations of the potent synthetic estrogen diethylstilbestrol (DES) induces abnormal development of the female reproductive tract via a mechanism that acts through the down-regulation of Wnt7a (wingless-type MMTV integration site family, member 7A). Diethylstilbestrol wingless-type MMTV integration site family, member 7A Mus musculus
2 Perinatal exposure to high concentrations of the potent synthetic estrogen diethylstilbestrol (DES) induces abnormal development of the female reproductive tract via a mechanism that acts through the down-regulation of Wnt7a (wingless-type MMTV integration site family, member 7A). Diethylstilbestrol wingless-type MMTV integration site family, member 7A Mus musculus
3 Perinatal exposure to high concentrations of the potent synthetic estrogen diethylstilbestrol (DES) induces abnormal development of the female reproductive tract via a mechanism that acts through the down-regulation of Wnt7a (wingless-type MMTV integration site family, member 7A). Diethylstilbestrol wingless-type MMTV integration site family, member 7A Mus musculus
4 Perinatal exposure to high concentrations of the potent synthetic estrogen diethylstilbestrol (DES) induces abnormal development of the female reproductive tract via a mechanism that acts through the down-regulation of Wnt7a (wingless-type MMTV integration site family, member 7A). Diethylstilbestrol wingless-type MMTV integration site family, member 7A Mus musculus