Title : Critical role of endogenous Akt/IAPs and MEK1/ERK pathways in counteracting endoplasmic reticulum stress-induced cell death.

Pub. Date : 2004 Nov 19

PMID : 15339911






1 Functional Relationships(s)
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1 Here, we have shown that endogenous Akt and ERK are rapidly activated and act as downstream effectors of phosphatidylinositol 3-kinase in thapsigargin- or tunicamycin-induced ER stress. Tunicamycin AKT serine/threonine kinase 1 Homo sapiens