Title : Leptin and insulin down-regulate angiopoietin-like protein 3, a plasma triglyceride-increasing factor.

Pub. Date : 2004 Sep 24

PMID : 15336575






5 Functional Relationships(s)
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Protein Name
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1 We reported previously that angiopoietin-like protein3 (ANGPTL3), a liver-specific secretory factor, increased plasma triglyceride (TG) via inhibition of lipoprotein lipase and free fatty acid (FFA) by activating adipose-lipolysis. Triglycerides angiopoietin-like 3 Mus musculus
2 We reported previously that angiopoietin-like protein3 (ANGPTL3), a liver-specific secretory factor, increased plasma triglyceride (TG) via inhibition of lipoprotein lipase and free fatty acid (FFA) by activating adipose-lipolysis. Triglycerides angiopoietin-like 3 Mus musculus
3 We reported previously that angiopoietin-like protein3 (ANGPTL3), a liver-specific secretory factor, increased plasma triglyceride (TG) via inhibition of lipoprotein lipase and free fatty acid (FFA) by activating adipose-lipolysis. Triglycerides angiopoietin-like 3 Mus musculus
4 We reported previously that angiopoietin-like protein3 (ANGPTL3), a liver-specific secretory factor, increased plasma triglyceride (TG) via inhibition of lipoprotein lipase and free fatty acid (FFA) by activating adipose-lipolysis. Triglycerides angiopoietin-like 3 Mus musculus
5 Our results suggest that elevated ANGPTL3 by leptin- or insulin-resistance is attributed to increased plasma TG and FFA concentrations in obesity. Triglycerides angiopoietin-like 3 Mus musculus