Title : 17beta-estradiol inhibits oxidative stress-induced apoptosis in keratinocytes by promoting Bcl-2 expression.

Pub. Date : 2003 Dec

PMID : 14675202






3 Functional Relationships(s)
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1 H-89, an inhibitor of cAMP-dependent protein kinase A, suppressed basal and 17beta-estradiol-induced cAMP response element-binding protein phosphorylation, cAMP response element-dependent transcriptional activity, Bcl-2 expression, and apoptosis resistance. N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide cathelicidin antimicrobial peptide Homo sapiens
2 H-89, an inhibitor of cAMP-dependent protein kinase A, suppressed basal and 17beta-estradiol-induced cAMP response element-binding protein phosphorylation, cAMP response element-dependent transcriptional activity, Bcl-2 expression, and apoptosis resistance. N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide cathelicidin antimicrobial peptide Homo sapiens
3 H-89, an inhibitor of cAMP-dependent protein kinase A, suppressed basal and 17beta-estradiol-induced cAMP response element-binding protein phosphorylation, cAMP response element-dependent transcriptional activity, Bcl-2 expression, and apoptosis resistance. N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide cathelicidin antimicrobial peptide Homo sapiens