Title : Increased iNOS activity is essential for pulmonary epithelial tight junction dysfunction in endotoxemic mice.

Pub. Date : 2004 Feb

PMID : 12896879






1 Functional Relationships(s)
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1 Treatment of endotoxemic mice with an isoform-selective iNOS inhibitor [l-N(6)-(1-iminoethyl)lysine; l-NIL] ameliorated LPS-induced changes in TJ protein expression and preserved bronchoalveolar epithelial barrier function. N(6)-(1-iminoethyl)lysine nitric oxide synthase 2, inducible Mus musculus