Title : Rapid increase in endothelial nitric oxide production by bradykinin is mediated by protein kinase A signaling pathway.

Pub. Date : 2003 Jul 11

PMID : 12821139






2 Functional Relationships(s)
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1 This acute effect of BK on both increases was blocked only by treatment of protein kinase A inhibitor H-89, but not by the inhibitors of calmodulin-dependent kinase II and protein kinase B, suggesting that the rapid increase in NO production by BK is mediated by the PKA-dependent phosphorylation of eNOS-Ser(1179). Serine kininogen 1 Bos taurus
2 This acute effect of BK on both increases was blocked only by treatment of protein kinase A inhibitor H-89, but not by the inhibitors of calmodulin-dependent kinase II and protein kinase B, suggesting that the rapid increase in NO production by BK is mediated by the PKA-dependent phosphorylation of eNOS-Ser(1179). Serine kininogen 1 Bos taurus