Title : Nitric oxide promotes p53 nuclear retention and sensitizes neuroblastoma cells to apoptosis by ionizing radiation.

Pub. Date : 2003 Apr

PMID : 12719724






2 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 An ATR-kinase dead mutant or caffeine, which blocks the kinase activity of ATR, effectively abolishes the ability of NO to cause p53 nuclear retention, concomitant with its inhibition of p53 serine 15 phosphorylation. Caffeine tumor protein p53 Homo sapiens
2 An ATR-kinase dead mutant or caffeine, which blocks the kinase activity of ATR, effectively abolishes the ability of NO to cause p53 nuclear retention, concomitant with its inhibition of p53 serine 15 phosphorylation. Caffeine tumor protein p53 Homo sapiens