Title : Fluticasone propionate downregulates nasal fibroblast functions involved in airway inflammation and remodeling.

Pub. Date : 2002 May

PMID : 12037401






2 Functional Relationships(s)
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1 Both bFGF-induced fibroblast proliferation and TNF-alpha-induced ICAM-1 expression were significantly reduced by fluticasone, starting at the dose of 1 and 10 nM, respectively (p < 0.05). Fluticasone tumor necrosis factor Homo sapiens
2 Fluticasone at concentrations of 1-100 nM effectively inhibited eotaxin release by TNF-alpha- or IL-4-stimulated fibroblasts (p < 0.05). Fluticasone tumor necrosis factor Homo sapiens