Title : Reduced perisomatic inhibition, increased excitatory transmission, and impaired long-term potentiation in mice deficient for the extracellular matrix glycoprotein tenascin-R.

Pub. Date : 2001 Jan

PMID : 11161481






1 Functional Relationships(s)
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1 A possible mechanism for increased excitatory synaptic transmission in mutants could involve modulation of inhibition, since TN-R and its associated carbohydrate HNK-1 decorate perisomatic interneurons. Carbohydrates beta-1,3-glucuronyltransferase 1 (glucuronosyltransferase P) Mus musculus