PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
20732396-1	2010	This work was designed to further study the mechanism by which sulforaphane (SFN) exerts a renoprotective effect against cisplatin (CIS)-induced damage.	sulforaphane	63-75	14-3-3 protein sigma	Sus scrofa	77-80
20732396-1	2010	This work was designed to further study the mechanism by which sulforaphane (SFN) exerts a renoprotective effect against cisplatin (CIS)-induced damage.	Cisplatin	121-130	14-3-3 protein sigma	Sus scrofa	77-80
20732396-4	2010	SFN effectively prevented the CIS-induced increase in reactive oxygen species (ROS) production and the decrease in NQO1 and gammaGCL activities and in glutathione (GSH) content.	Reactive Oxygen Species	54-77	14-3-3 protein sigma	Sus scrofa	0-3
20732396-4	2010	SFN effectively prevented the CIS-induced increase in reactive oxygen species (ROS) production and the decrease in NQO1 and gammaGCL activities and in glutathione (GSH) content.	Reactive Oxygen Species	79-82	14-3-3 protein sigma	Sus scrofa	0-3
20732396-4	2010	SFN effectively prevented the CIS-induced increase in reactive oxygen species (ROS) production and the decrease in NQO1 and gammaGCL activities and in glutathione (GSH) content.	Glutathione	151-162	14-3-3 protein sigma	Sus scrofa	0-3
20732396-4	2010	SFN effectively prevented the CIS-induced increase in reactive oxygen species (ROS) production and the decrease in NQO1 and gammaGCL activities and in glutathione (GSH) content.	Glutathione	164-167	14-3-3 protein sigma	Sus scrofa	0-3
20732396-5	2010	The protective effect of SFN on ROS production and cell viability was prevented by buthionine sulfoximine (BSO), an inhibitor of gammaGCL, and by dicoumarol, an inhibitor of NQO1.	Reactive Oxygen Species	32-35	14-3-3 protein sigma	Sus scrofa	25-28
20732396-5	2010	The protective effect of SFN on ROS production and cell viability was prevented by buthionine sulfoximine (BSO), an inhibitor of gammaGCL, and by dicoumarol, an inhibitor of NQO1.	Buthionine Sulfoximine	83-105	14-3-3 protein sigma	Sus scrofa	25-28
20732396-5	2010	The protective effect of SFN on ROS production and cell viability was prevented by buthionine sulfoximine (BSO), an inhibitor of gammaGCL, and by dicoumarol, an inhibitor of NQO1.	Buthionine Sulfoximine	107-110	14-3-3 protein sigma	Sus scrofa	25-28
20732396-5	2010	The protective effect of SFN on ROS production and cell viability was prevented by buthionine sulfoximine (BSO), an inhibitor of gammaGCL, and by dicoumarol, an inhibitor of NQO1.	gammagcl	129-137	14-3-3 protein sigma	Sus scrofa	25-28
20732396-5	2010	The protective effect of SFN on ROS production and cell viability was prevented by buthionine sulfoximine (BSO), an inhibitor of gammaGCL, and by dicoumarol, an inhibitor of NQO1.	Dicumarol	146-156	14-3-3 protein sigma	Sus scrofa	25-28
20732396-6	2010	SFN was also able to prevent the CIS-induced mitochondrial alterations both in LLC-PK1 cells (loss of membrane potential) and in isolated mitochondria (inhibition of mitochondrial calcium uptake, release of cytochrome c, and decrease in GSH content, aconitase activity, adenosine triphosphate (ATP) content and oxygen consumption).	Calcium	180-187	14-3-3 protein sigma	Sus scrofa	0-3
20732396-6	2010	SFN was also able to prevent the CIS-induced mitochondrial alterations both in LLC-PK1 cells (loss of membrane potential) and in isolated mitochondria (inhibition of mitochondrial calcium uptake, release of cytochrome c, and decrease in GSH content, aconitase activity, adenosine triphosphate (ATP) content and oxygen consumption).	Glutathione	237-240	14-3-3 protein sigma	Sus scrofa	0-3
20732396-6	2010	SFN was also able to prevent the CIS-induced mitochondrial alterations both in LLC-PK1 cells (loss of membrane potential) and in isolated mitochondria (inhibition of mitochondrial calcium uptake, release of cytochrome c, and decrease in GSH content, aconitase activity, adenosine triphosphate (ATP) content and oxygen consumption).	Adenosine Triphosphate	270-292	14-3-3 protein sigma	Sus scrofa	0-3
20732396-6	2010	SFN was also able to prevent the CIS-induced mitochondrial alterations both in LLC-PK1 cells (loss of membrane potential) and in isolated mitochondria (inhibition of mitochondrial calcium uptake, release of cytochrome c, and decrease in GSH content, aconitase activity, adenosine triphosphate (ATP) content and oxygen consumption).	Adenosine Triphosphate	294-297	14-3-3 protein sigma	Sus scrofa	0-3
20732396-6	2010	SFN was also able to prevent the CIS-induced mitochondrial alterations both in LLC-PK1 cells (loss of membrane potential) and in isolated mitochondria (inhibition of mitochondrial calcium uptake, release of cytochrome c, and decrease in GSH content, aconitase activity, adenosine triphosphate (ATP) content and oxygen consumption).	Oxygen	311-317	14-3-3 protein sigma	Sus scrofa	0-3