PMID-sentid Pub_year Sent_text comp_official_name comp_offset protein_name organism prot_offset 20127209-0 2010 Sequence-specific 1H, 13C, and 15N resonance assignments of Diva (Boo), an apoptosis regulator of the Bcl-2 family. Hydrogen 18-20 Bcl2-like 10 Mus musculus 60-64 21171085-4 2011 The methylation status of the BCL2L10 gene promoter was examined by bisulphite sequencing in fresh gastric normal and carcinoma tissues. hydrogen sulfite 68-78 Bcl2-like 10 Mus musculus 30-37 20127209-0 2010 Sequence-specific 1H, 13C, and 15N resonance assignments of Diva (Boo), an apoptosis regulator of the Bcl-2 family. Hydrogen 18-20 Bcl2-like 10 Mus musculus 66-69 20127209-0 2010 Sequence-specific 1H, 13C, and 15N resonance assignments of Diva (Boo), an apoptosis regulator of the Bcl-2 family. 13c 22-25 Bcl2-like 10 Mus musculus 60-64 20127209-0 2010 Sequence-specific 1H, 13C, and 15N resonance assignments of Diva (Boo), an apoptosis regulator of the Bcl-2 family. 13c 22-25 Bcl2-like 10 Mus musculus 66-69 20127209-0 2010 Sequence-specific 1H, 13C, and 15N resonance assignments of Diva (Boo), an apoptosis regulator of the Bcl-2 family. 15n 31-34 Bcl2-like 10 Mus musculus 60-64 20127209-0 2010 Sequence-specific 1H, 13C, and 15N resonance assignments of Diva (Boo), an apoptosis regulator of the Bcl-2 family. 15n 31-34 Bcl2-like 10 Mus musculus 66-69 19439730-8 2009 We concluded that the role of Bcl2l10 is strongly associated with oocyte maturation, especially at the MI-MII transition. Methicillin 106-109 Bcl2-like 10 Mus musculus 30-37 34086879-8 2021 IAA exposure decreased expression of the pro-apoptotic factor Bok, the cell cycle regulator Ccnd2, and borderline decreased expression of the anti-apoptotic factor Bcl2l10, the pro-apoptotic factor Aimf1, and the steroidogenic factor Cyp19a1 compared to control. Iodoacetic Acid 0-3 Bcl2-like 10 Mus musculus 164-171 19154466-1 2009 OBJECTIVE: To investigate if sildenafil citrate can inhibit the functional and structural changes of the detrusor in a murine model of bladder outlet obstruction (BOO). Sildenafil Citrate 29-47 Bcl2-like 10 Mus musculus 163-166 19154466-11 2009 BOO caused an increase in Pdet compared to controls, with a mean (SD) of 25 (7) vs 12 (5) cm H2O. Water 93-96 Bcl2-like 10 Mus musculus 0-3 19154466-14 2009 BOO was associated with a greater bladder weight than in control mice, with a mean (SD) of 89 (32) vs 27 (6) mg (P = 0.001), which was decreased with sildenafil treatment, to 40 (14) vs 89 (32) mg (P = 0.013). Sildenafil Citrate 150-160 Bcl2-like 10 Mus musculus 0-3 19154466-15 2009 BOO caused an increase in detrusor muscular hypertrophy vs control mice, with a median H&E score of 3 vs 2 (P = 0.01) and an increase in fibrosis vs control mice, with a median trichrome score of 3 vs 2 (P = 0.01). Adenosine Monophosphate 89-92 Bcl2-like 10 Mus musculus 0-3 19154466-19 2009 Treatment with 6 weeks of oral sildenafil beginning at the time of BOO prevented the increase in detrusor overactivity without affecting voiding pressures, and prevented the increase in detrusor muscle hypertrophy and collagen deposition that otherwise occurred with BOO. Sildenafil Citrate 31-41 Bcl2-like 10 Mus musculus 67-70 19154466-19 2009 Treatment with 6 weeks of oral sildenafil beginning at the time of BOO prevented the increase in detrusor overactivity without affecting voiding pressures, and prevented the increase in detrusor muscle hypertrophy and collagen deposition that otherwise occurred with BOO. Sildenafil Citrate 31-41 Bcl2-like 10 Mus musculus 267-270 34086879-9 2021 IAA exposure increased expression of the pro-apoptotic factors Bax and Aimf1, the anti-apoptotic factor Bcl2l10, the cell cycle regulators Ccna2, Ccnb1, Ccne1, and Cdk4, and estrogen receptor Esr1 compared to control. Iodoacetic Acid 0-3 Bcl2-like 10 Mus musculus 104-111