PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
25120693-2	2014	The present study aimed to investigate the association between X-ray repair complementing defective repair in Chinese hamster cells 2 (XRCC2) SNPs and colorectal cancer (CRC) cell sensitivity to the poly(ADP-ribose) polymerase (PARP) 1 inhibitor olaparib (AZD2281).	olaparib	246-254	DNA repair protein XRCC2	Cricetulus griseus	135-140
25120693-2	2014	The present study aimed to investigate the association between X-ray repair complementing defective repair in Chinese hamster cells 2 (XRCC2) SNPs and colorectal cancer (CRC) cell sensitivity to the poly(ADP-ribose) polymerase (PARP) 1 inhibitor olaparib (AZD2281).	olaparib	256-263	DNA repair protein XRCC2	Cricetulus griseus	135-140
22302683-7	2012	In contrast, we confirm that the V79 xrcc2 mutant does show increased sensitivity to hydroxyurea under some conditions, which was correlated to its attenuated RAD51 focus response.	Hydroxyurea	85-96	DNA repair protein XRCC2	Cricetulus griseus	37-42
15782136-3	2005	V79-SMRAD51 cells are sensitive to mitomycin C (MMC), but SMRAD51 did not further sensitize irs1 cells to MMC, showing that SMRAD51 and XRCC2 act on the same pathway for resistance to MMC.	Mitomycin	35-46	DNA repair protein XRCC2	Cricetulus griseus	136-141
15782136-3	2005	V79-SMRAD51 cells are sensitive to mitomycin C (MMC), but SMRAD51 did not further sensitize irs1 cells to MMC, showing that SMRAD51 and XRCC2 act on the same pathway for resistance to MMC.	Mitomycin	48-51	DNA repair protein XRCC2	Cricetulus griseus	136-141
14737117-1	2004	We recently reported that two Chinese hamster mutants deficient in the RAD51 paralogs XRCC2 and XRCC3 show reduced radiosensitization after treatment with caffeine, thus implicating homology-directed repair (HDR) of DNA double-strand breaks (DSBs) in the mechanism of caffeine radiosensitization.	Caffeine	155-163	DNA repair protein XRCC2	Cricetulus griseus	86-91
14737117-1	2004	We recently reported that two Chinese hamster mutants deficient in the RAD51 paralogs XRCC2 and XRCC3 show reduced radiosensitization after treatment with caffeine, thus implicating homology-directed repair (HDR) of DNA double-strand breaks (DSBs) in the mechanism of caffeine radiosensitization.	Caffeine	268-276	DNA repair protein XRCC2	Cricetulus griseus	86-91
12584174-4	2003	In the work reported here we investigated the possible role of the Rad51-like HR proteins XRCC2, XRCC3 and Rad51C in apoptosis following the induction of DSBs by camptothecin.	dsbs	154-158	DNA repair protein XRCC2	Cricetulus griseus	90-95
12584174-4	2003	In the work reported here we investigated the possible role of the Rad51-like HR proteins XRCC2, XRCC3 and Rad51C in apoptosis following the induction of DSBs by camptothecin.	Camptothecin	162-174	DNA repair protein XRCC2	Cricetulus griseus	90-95
12202770-8	2002	Surprisingly, XRCC2 and XRCC3 cells are defective in the uncoupling step of cisplatin interstrand cross-link repair, suggesting that homologous recombination might be initiated prior to excision of this type of cross-link.	Cisplatin	76-85	DNA repair protein XRCC2	Cricetulus griseus	14-19
24570594-0	2014	miR-7 inhibits colorectal cancer cell proliferation and induces apoptosis by targeting XRCC2.	mir-7	0-5	DNA repair protein XRCC2	Cricetulus griseus	87-92
18519695-10	2008	irs1 and VC8 cells defective in homologous recombination (HR) due to inactivation of XRCC2 and BRCA2, respectively, were more sensitive to artesunate than the corresponding wild-type.	Artesunate	139-149	DNA repair protein XRCC2	Cricetulus griseus	85-90