PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
25933513-7	2015	Using PSF1a-overexpressing NIH-3T3 cells in which the endogenous PSF1 promoter is suppressed, we found that TSA treatment promotes production of the shorter form of PSF1 possibly by inducing recruitment of E2F family factors upstream of the PSF1 transcription start site.	trichostatin A	108-111	GINS complex subunit 1 (Psf1 homolog)	Mus musculus	6-10
25933513-7	2015	Using PSF1a-overexpressing NIH-3T3 cells in which the endogenous PSF1 promoter is suppressed, we found that TSA treatment promotes production of the shorter form of PSF1 possibly by inducing recruitment of E2F family factors upstream of the PSF1 transcription start site.	trichostatin A	108-111	GINS complex subunit 1 (Psf1 homolog)	Mus musculus	65-69
25933513-7	2015	Using PSF1a-overexpressing NIH-3T3 cells in which the endogenous PSF1 promoter is suppressed, we found that TSA treatment promotes production of the shorter form of PSF1 possibly by inducing recruitment of E2F family factors upstream of the PSF1 transcription start site.	trichostatin A	108-111	GINS complex subunit 1 (Psf1 homolog)	Mus musculus	65-69
18984863-5	2009	Whereas young PSF1(+/-) mutant mice develop normally, are fertile, and have no obvious differences in hematopoiesis in the steady state compared with wild-type mice, intravenous injection of 5-fluorouracil (5-FU) is lethal in PSF1(+/-) mice, resulting from a delay in induction of HSC proliferation during ablated BM reconstitution.	Fluorouracil	191-205	GINS complex subunit 1 (Psf1 homolog)	Mus musculus	226-230
24244394-5	2013	We have previously reported that haploinsufficiency of PSF1 resulted in failure of acute proliferation of bone marrow hematopoietic stem cells (HSCs) during reconstitution of bone marrow ablated by 5-FU treatment.	Fluorouracil	198-202	GINS complex subunit 1 (Psf1 homolog)	Mus musculus	55-59
34414190-8	2021	Moreover, knocking down endogenous GINS1 with shGINS1 increased the sensitivity of HCC cells to sorafenib, and restoring HRAS or stem associated pathway partly recovered the sorafenib resistance.	Sorafenib	96-105	GINS complex subunit 1 (Psf1 homolog)	Mus musculus	35-40
34414190-8	2021	Moreover, knocking down endogenous GINS1 with shGINS1 increased the sensitivity of HCC cells to sorafenib, and restoring HRAS or stem associated pathway partly recovered the sorafenib resistance.	Sorafenib	174-183	GINS complex subunit 1 (Psf1 homolog)	Mus musculus	35-40
34414190-9	2021	Overall, the collective findings highlight GINS1 functions in hepatocarcinogenesis and sorafenib resistance, and indicate its potential use of GINS1 in drug-resistant HCC.	Sorafenib	87-96	GINS complex subunit 1 (Psf1 homolog)	Mus musculus	43-48