PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
2324687-2	1990	To study this problem the immunogenic MHC class I antigen, K216 was transfected into a progressor tumor.	C15H21NO2S	59-63	H2-T3-like	Mus musculus	38-57
23451104-0	2013	Suppression of the macrophage proteasome by ethanol impairs MHC class I antigen processing and presentation.	Ethanol	44-51	H2-T3-like	Mus musculus	60-79
3359976-3	1988	Monoclonal antibodies against MHC class I antigen were able to stimulate testosterone production in mouse Leydig cells with the same potency as LH.	Testosterone	73-85	H2-T3-like	Mus musculus	30-49
3964822-1	1985	Oligonucleotide-directed, site-specific mutagenesis has been employed to elucidate the role of individual amino acids on the expression and function of a MHC class I antigen.	Oligonucleotides	0-15	H2-T3-like	Mus musculus	154-173
23284054-0	2013	TLR7 triggering with polyuridylic acid promotes cross-presentation in CD8alpha+ conventional dendritic cells by enhancing antigen preservation and MHC class I antigen permanence on the dendritic cell surface.	Poly U	21-38	H2-T3-like	Mus musculus	147-166
16341058-5	2006	Expression of the allogeneic MHC class I antigen on bone marrow-derived cells following transplantation of Diprotin A-treated cells was sufficient to induce transplantation tolerance.	diprotin A	107-117	H2-T3-like	Mus musculus	29-48
18645033-3	2008	We identified three novel mouse TH (mTH3) derived peptides with high predicted binding affinity to MHC class I antigen H2-K(k) according to the prediction program SYFPEITHI and computer modeling of epitopes into the MHC class I antigen binding groove.	mth3	36-40	H2-T3-like	Mus musculus	99-118
18645033-3	2008	We identified three novel mouse TH (mTH3) derived peptides with high predicted binding affinity to MHC class I antigen H2-K(k) according to the prediction program SYFPEITHI and computer modeling of epitopes into the MHC class I antigen binding groove.	mth3	36-40	H2-T3-like	Mus musculus	216-235
18455279-3	2008	In this study, we analyzed the effect of liposomal lipid A [L(LA)] on the MHC class I antigen processing machinery in murine antigen presenting cells (APCs).	Lipid A	51-58	H2-T3-like	Mus musculus	74-93
17786443-3	2008	Here, we tested our hypothesis that suppression of TAMs can be achieved in syngeneic BALB/c mice with oral minigene vaccines against murine MHC class I antigen epitopes of Legumain, an asparaginyl endopeptidase and a member of the C13 family of cystine proteases which is overexpressed on TAMs in the tumor stroma.	tams	51-55	H2-T3-like	Mus musculus	140-159
11888922-5	2002	Inhibition of sensitization by the addition of cycloheximide and brefeldin A to the culture indicated the requirement of newly generated MHC class I antigen molecules and the involvement of transport of the peptide MHC class I complex from the endoplasmic reticulum to the Golgi.	Cycloheximide	47-60	H2-T3-like	Mus musculus	137-156
11888922-5	2002	Inhibition of sensitization by the addition of cycloheximide and brefeldin A to the culture indicated the requirement of newly generated MHC class I antigen molecules and the involvement of transport of the peptide MHC class I complex from the endoplasmic reticulum to the Golgi.	Brefeldin A	65-76	H2-T3-like	Mus musculus	137-156