PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
6546848-6	1984	Cross-reactivity of AFB1-diol antibody in the competitive ELISA with AF analogs was as follows: AFB1-diol, 100%; AFB1, 200%; AFM1, 130%; AFB2a, 100%; AFG1, 6%; AFG2, 4%; aflatoxicol, 20%; AFQ1, 2%; AFB1-modified DNA, 32%; and 2,3-dihydro-2-(N7-guanyl)-3-hydroxy AFB1, 0.6%.	afb1-diol	20-29	AFG1 like ATPase	Homo sapiens	150-154
2519710-2	1989	Human liver microsomal cytochrome P-450NF oxidized the dihydrofurans (in the presence of calf thymus DNA) to give guanyl-N7 adducts in the order AFB1 greater than STG greater than AFG1.	dihydrofurans	55-68	AFG1 like ATPase	Homo sapiens	180-184
2519710-3	1989	The order of the umu response seen was STG greater than AFB1 greater than AFG1, when either the dihydrofurans were activated enzymatically or the synthetic epoxides of the dihydrofurans were added directly to the bacteria.	dihydrofurans	96-109	AFG1 like ATPase	Homo sapiens	74-78
2519710-3	1989	The order of the umu response seen was STG greater than AFB1 greater than AFG1, when either the dihydrofurans were activated enzymatically or the synthetic epoxides of the dihydrofurans were added directly to the bacteria.	dihydrofurans	172-185	AFG1 like ATPase	Homo sapiens	74-78
30952310-3	2019	Under optimized conditions (1.25 mL of 5 mg L-1 MIP-PEG-ZnS QDs solution, pH 5.0, and 12 min delay time before scanning), the prepared MIP-QDs composite was found to offer high affinity and selectivity for AFs (AFB1, AFB2, AFG1 and AFG2).	Zinc	56-59	AFG1 like ATPase	Homo sapiens	223-227
32983001-2	2020	Aflatoxin B1 (AFB1) and aflatoxin B2 (AFB2) as well as aflatoxin G1(AFG1) and aflatoxin G2 (AFG2) occur in the contaminated foods and feed.	aflatoxin G1	55-67	AFG1 like ATPase	Homo sapiens	68-72
31793394-5	2020	Aflatoxin B1was found in all positive samples and co-occurred with AFB2, AFG1, and AFG2.	Aflatoxins	0-9	AFG1 like ATPase	Homo sapiens	73-77
31814038-0	2019	An amino-functionalized zirconium-based metal-organic framework of type UiO-66-NH2 covered with a molecularly imprinted polymer as a sorbent for the extraction of aflatoxins AFB1, AFB2, AFG1 and AFG2 from grain.	Zirconium	24-33	AFG1 like ATPase	Homo sapiens	186-190
31814038-0	2019	An amino-functionalized zirconium-based metal-organic framework of type UiO-66-NH2 covered with a molecularly imprinted polymer as a sorbent for the extraction of aflatoxins AFB1, AFB2, AFG1 and AFG2 from grain.	Metals	40-45	AFG1 like ATPase	Homo sapiens	186-190
31814038-0	2019	An amino-functionalized zirconium-based metal-organic framework of type UiO-66-NH2 covered with a molecularly imprinted polymer as a sorbent for the extraction of aflatoxins AFB1, AFB2, AFG1 and AFG2 from grain.	Polymers	120-127	AFG1 like ATPase	Homo sapiens	186-190
32517894-6	2020	Aflatoxins were detected in a maximum of 13% (AFB1), 16% (AFB2), 1% (AFG1), 2% (AFG2) and 19% (AFM1) of the urine samples.	Aflatoxins	0-10	AFG1 like ATPase	Homo sapiens	69-73
30468840-4	2019	Basic AFs (AFB1, AFB2, AFG1, and AFG2) are metabolized in the liver or by microbes that produce hydroxylated metabolites (AFM1, AFM2, and AFP1) and aflatoxicol (AFL), soluble in water and easy to dispose.	aflatoxicol	148-159	AFG1 like ATPase	Homo sapiens	23-27
30478833-1	2019	Aflatoxin G1 (AFG1 ), a member of the AF family with cytotoxic and carcinogenic properties, could cause DNA damage in alveolar type II (AT-II) cells and induce lung adenocarcinoma.	aflatoxin G1	0-12	AFG1 like ATPase	Homo sapiens	14-18
30832301-6	2019	Trace element analysis showed that the concentrations of several metal ions differed by factors of >100, and the carbons that most effectively increased AFG1 production contained higher amounts of metal ions.	Carbon	113-120	AFG1 like ATPase	Homo sapiens	153-157
30832301-6	2019	Trace element analysis showed that the concentrations of several metal ions differed by factors of >100, and the carbons that most effectively increased AFG1 production contained higher amounts of metal ions.	Metals	197-202	AFG1 like ATPase	Homo sapiens	153-157
30832301-7	2019	Adding 5 mg L-1 Fe or Mg ions increased AFG1 production even without activated carbon.	Iron	16-18	AFG1 like ATPase	Homo sapiens	40-44
30832301-7	2019	Adding 5 mg L-1 Fe or Mg ions increased AFG1 production even without activated carbon.	Magnesium	22-24	AFG1 like ATPase	Homo sapiens	40-44
30832301-8	2019	Furthermore, co-addition of both ions increased AFG1 production stably with the addition of carbon.	Carbon	92-98	AFG1 like ATPase	Homo sapiens	48-52
30468840-4	2019	Basic AFs (AFB1, AFB2, AFG1, and AFG2) are metabolized in the liver or by microbes that produce hydroxylated metabolites (AFM1, AFM2, and AFP1) and aflatoxicol (AFL), soluble in water and easy to dispose.	aflatoxicol	161-164	AFG1 like ATPase	Homo sapiens	23-27
30468840-4	2019	Basic AFs (AFB1, AFB2, AFG1, and AFG2) are metabolized in the liver or by microbes that produce hydroxylated metabolites (AFM1, AFM2, and AFP1) and aflatoxicol (AFL), soluble in water and easy to dispose.	Water	178-183	AFG1 like ATPase	Homo sapiens	23-27
28844113-11	2017	We hypothesize that the formation of  OH radicals initiated by UV light may have caused photolysis of AFB1, AFB2, and AFG1 molecules.	oh radicals	38-49	AFG1 like ATPase	Homo sapiens	118-122
28911616-5	2016	After screening, five MAbs (AFM1-1, AFM1-3, AFM1-9, AFM1-11, and AFM1-17) were obtained that showed cross-reactivity with aflatoxin B1 (AFB1) and aflatoxin G1 (AFG1) but with no other tested compounds.	Aflatoxin B1	122-134	AFG1 like ATPase	Homo sapiens	160-164
28801561-2	2017	Our previous study showed Aflatoxin G1 (AFG1) could induce lung adenocarcinoma in mice.	Aflatoxins	26-35	AFG1 like ATPase	Homo sapiens	40-44
25336278-1	2015	Recently, we discovered that Aflatoxin G1 (AFG1 ) induces chronic lung inflammatory responses, which may contribute to lung tumorigenesis in Balb/C mice.	aflatoxin G1	29-41	AFG1 like ATPase	Homo sapiens	43-47
27187470-2	2016	We developed a murine monoclonal antibody of immunoglobulin A (IgA) isotype with a strong binding affinity to aflatoxin B1 (AFB1), aflatoxin B2 (AFB2), aflatoxin G1 (AFG1), aflatoxin G2 (AFG2) and aflatoxin M1 (AFM1).	Aflatoxin B1	110-122	AFG1 like ATPase	Homo sapiens	166-170
27323408-5	2016	LACE1 physically interacts with p53 and is necessary for mitomycin c-induced translocation of p53 into mitochondria.	Mitomycin	57-68	AFG1 like ATPase	Homo sapiens	0-5
26133228-4	2015	The aflatoxin most frequently detected in the samples was AFG1, present in 57.9% of samples, while the other aflatoxins were rarely present.	Aflatoxins	4-13	AFG1 like ATPase	Homo sapiens	58-62
25603600-5	2014	The transmicivities of AFB1, AFG1 and AFG2 reached the highest at the incubation time of 60 minutes but 120 minutes for DON and AFB2.	DONS	120-123	AFG1 like ATPase	Homo sapiens	29-33
25606005-3	2014	Aflatoxins were found in 124 out of the 729 analyzed samples: 65 containing aflatoxin B1 (AFB1), 24 with aflatoxin B2 (AFB2), 15 with aflatoxin G1 (AFG1), and 20 samples with aflatoxin G2 (AFG2).	Aflatoxins	0-10	AFG1 like ATPase	Homo sapiens	148-152
25603600-5	2014	The transmicivities of AFB1, AFG1 and AFG2 reached the highest at the incubation time of 60 minutes but 120 minutes for DON and AFB2.	aflatoxin B2	128-132	AFG1 like ATPase	Homo sapiens	29-33
22971039-1	2012	A simple method for the reduction of aflatoxins B1 (AFB1), B2 (AFB2), G1 (AFG1), G2 (AFG2) and ochratoxin A (OTA) in white pepper was studied.	Aflatoxins	37-47	AFG1 like ATPase	Homo sapiens	74-78
25029403-3	2014	A positive correlation was found between four types of aflatoxins in all the tested samples (p < 0.01) and the positive correlation between AFG1 and AFG2 was significant (r(2) = 0.708).	Aflatoxins	55-65	AFG1 like ATPase	Homo sapiens	143-147
24090735-5	2013	Thus, we explore whether AFG1 activates the reactive oxygen species (ROS)/MAPK/apoptosis pathway to cause cell damage in human AT-II cells like the cell line (A549).	Reactive Oxygen Species	44-67	AFG1 like ATPase	Homo sapiens	25-29
24090735-5	2013	Thus, we explore whether AFG1 activates the reactive oxygen species (ROS)/MAPK/apoptosis pathway to cause cell damage in human AT-II cells like the cell line (A549).	Reactive Oxygen Species	69-72	AFG1 like ATPase	Homo sapiens	25-29
24090735-6	2013	We found AFG1 induced oxidative stress by increasing ROS generation and caused DNA double-strand breaks (DSBs) by up-regulating gammaH2AX expression.	Reactive Oxygen Species	53-56	AFG1 like ATPase	Homo sapiens	9-13
24090735-8	2013	Pre-treatment with antioxidant n-acetyl-l-cysteine (NAC) reduced ROS generation and DNA DSBs, inhibited apoptosis, and increased cell viability in AFG1-treated cells.	Acetylcysteine	31-50	AFG1 like ATPase	Homo sapiens	147-151
24090735-8	2013	Pre-treatment with antioxidant n-acetyl-l-cysteine (NAC) reduced ROS generation and DNA DSBs, inhibited apoptosis, and increased cell viability in AFG1-treated cells.	dsbs	88-92	AFG1 like ATPase	Homo sapiens	147-151
24090735-9	2013	Furthermore, we found AFG1 activated ROS-mediated JNK and p38 pathways to induce cell apoptosis in A549 cells.	Reactive Oxygen Species	37-40	AFG1 like ATPase	Homo sapiens	22-26
24090735-10	2013	In conclusion, our results indicate that AFG1 induces oxidative DNA damage and triggers apoptosis through ROS-mediated JNK and p38 signaling pathways in A549 cells, which may contribute to AFG1-induced AT-II cell damage.	Reactive Oxygen Species	106-109	AFG1 like ATPase	Homo sapiens	41-45
24090735-10	2013	In conclusion, our results indicate that AFG1 induces oxidative DNA damage and triggers apoptosis through ROS-mediated JNK and p38 signaling pathways in A549 cells, which may contribute to AFG1-induced AT-II cell damage.	Reactive Oxygen Species	106-109	AFG1 like ATPase	Homo sapiens	189-193
23907605-6	2013	In addition, AFG1 increased 8-OHdG and gammaH2AX in the nuclies and induced S phase arrest and DNA damage in B-2A13 cells, and the proteins related to DNA damage responses, such as ATM, ATR, Chk2, p53, BRCA1, and gammaH2AX, were activated.	8-ohdg	28-34	AFG1 like ATPase	Homo sapiens	13-17
23907605-7	2013	All the above effects were inhibited by nicotine (a substrate of CYP2A13) or 8-MOP (an inhibitor of CYP enzymes), confirming that CYP2A13 mediated the AFG1-induced cytotoxicity and DNA damages.	Nicotine	40-48	AFG1 like ATPase	Homo sapiens	151-155
23907605-7	2013	All the above effects were inhibited by nicotine (a substrate of CYP2A13) or 8-MOP (an inhibitor of CYP enzymes), confirming that CYP2A13 mediated the AFG1-induced cytotoxicity and DNA damages.	Methoxsalen	77-82	AFG1 like ATPase	Homo sapiens	151-155
21957681-9	2011	Aflatoxin metabolites, apart from AFM1 and AFB1 present in the weaning foods, were AFG1 and AFG2.	Aflatoxins	0-9	AFG1 like ATPase	Homo sapiens	83-87
11938994-5	1999	The lymphocytes treated with DON and AFG1 showed characteristic "ladder" pattern in agarose gel electrophoresis.	Sepharose	84-91	AFG1 like ATPase	Homo sapiens	37-41
12217366-1	2002	The kinetics of reduction of AFB(1) to AFB(2) and AFG(1) to AFG(2) by lactic acid has been investigated in dilute aqueous acidic solutions (pH 3.35-4.50) as a function of the concentrations of lactic acid, AFB(1), AFG(1) and hydrogen ion at 37 degrees C. The rate of the reaction was found to be first order with respect to the concentrations of lactic acid and aflatoxins and independent on hydrogen ion concentration.	Lactic Acid	70-81	AFG1 like ATPase	Homo sapiens	50-56
12217366-1	2002	The kinetics of reduction of AFB(1) to AFB(2) and AFG(1) to AFG(2) by lactic acid has been investigated in dilute aqueous acidic solutions (pH 3.35-4.50) as a function of the concentrations of lactic acid, AFB(1), AFG(1) and hydrogen ion at 37 degrees C. The rate of the reaction was found to be first order with respect to the concentrations of lactic acid and aflatoxins and independent on hydrogen ion concentration.	Lactic Acid	70-81	AFG1 like ATPase	Homo sapiens	214-220
12217366-3	2002	The proposed mechanisms involve an overall transfer of two protons and two electrons from lactic acid to AFB(1) and AFG(1) to give the corresponding reduced less toxic products AFB(2) and AFG(2) and the oxidised product pyruvic acid.	Lactic Acid	90-101	AFG1 like ATPase	Homo sapiens	116-122
12217366-3	2002	The proposed mechanisms involve an overall transfer of two protons and two electrons from lactic acid to AFB(1) and AFG(1) to give the corresponding reduced less toxic products AFB(2) and AFG(2) and the oxidised product pyruvic acid.	afb	177-180	AFG1 like ATPase	Homo sapiens	116-122
12217366-3	2002	The proposed mechanisms involve an overall transfer of two protons and two electrons from lactic acid to AFB(1) and AFG(1) to give the corresponding reduced less toxic products AFB(2) and AFG(2) and the oxidised product pyruvic acid.	Pyruvic Acid	220-232	AFG1 like ATPase	Homo sapiens	116-122
12131971-6	2002	Peanuts and its products showed the highest levels of aflatoxin contamination (34.7%) with up to 1280 mg/kg of AFB1+AFG1 and 1706 mg/kg of total aflatoxins.	Aflatoxins	54-63	AFG1 like ATPase	Homo sapiens	116-120
11829670-6	2002	The relative cross-reactivity with aflatoxins (AF) and ochratoxin A, assessed as the amount of AFM1 necessary to cause 50% inhibition of binding, was 5% for AFB1 and much less for AFB2, AFG1, and AFG2; there was no reaction with ochratoxin A.	Aflatoxins	35-45	AFG1 like ATPase	Homo sapiens	186-190
20630912-0	2011	Protective effects of selenium against sister chromatid exchange induced by AFG 1 in human lymphocytes in vitro.	Selenium	22-30	AFG1 like ATPase	Homo sapiens	76-81
24779628-1	2010	Aflatoxins (AFB1, AFB2, AFG1 and AFG2) are immunosuppressant, mutagenic, teratogenic and carcinogenic agents with a widespread presence in foodstuffs.	Aflatoxins	0-10	AFG1 like ATPase	Homo sapiens	24-28
12244755-3	2002	RESULTS: DNA agarose gel electrophoresis results showed the characteristic "ladder" pattern of apoptosis in HPBLs treated with ST, DON and AFG1.	Sepharose	13-20	AFG1 like ATPase	Homo sapiens	139-143
11829670-6	2002	The relative cross-reactivity with aflatoxins (AF) and ochratoxin A, assessed as the amount of AFM1 necessary to cause 50% inhibition of binding, was 5% for AFB1 and much less for AFB2, AFG1, and AFG2; there was no reaction with ochratoxin A.	ochratoxin A	55-67	AFG1 like ATPase	Homo sapiens	186-190
11765082-4	2001	Chromatographic responses of AFB1 and AFG1 achieved using beta-CD dissolved in the mobile phase were enhanced, respectively, 8 and 12 times, and 10 and 15 times with beta-CD-Su.	betadex	58-65	AFG1 like ATPase	Homo sapiens	38-42
11765082-4	2001	Chromatographic responses of AFB1 and AFG1 achieved using beta-CD dissolved in the mobile phase were enhanced, respectively, 8 and 12 times, and 10 and 15 times with beta-CD-Su.	beta-cd-su	166-176	AFG1 like ATPase	Homo sapiens	38-42
35594247-7	2022	Then, 30 CF samples were collected and the level of aflatoxins (AFB1, AFB2, AFG1, and AFG2) was determined using a validated method.	Aflatoxins	52-62	AFG1 like ATPase	Homo sapiens	76-80
1899057-0	1991	Identification of an aflatoxin G1-serum albumin adduct and its relevance to the measurement of human exposure to aflatoxins.	Aflatoxins	113-123	AFG1 like ATPase	Homo sapiens	21-33
1899057-2	1991	The product isolated from a Pronase digest of in vivo-modified albumin was identical by chromatographic retention time to the synthetic product obtained by the acylase-catalysed deacetylation product of N alpha-acetyl-L-lysine with 8,9-dihydro-8,9-dibromo-AFG1.	N(alpha)-acetyllysine	203-226	AFG1 like ATPase	Homo sapiens	256-260
1899057-3	1991	The in vitro product, AFG1-lysine, was characterized by UV, fluorescence, 1H- and 13C-NMR spectroscopy and fast atom bombardment MS. A competitive enzyme-linked immunoassay for this adduct was established using polyclonal antibodies to AFB1 and this was used together with an HPLC-fluorescence technique to quantitate the in vivo formation of AFG1-albumin adducts in comparison to AFB1.	Lysine	27-33	AFG1 like ATPase	Homo sapiens	22-26
1899057-3	1991	The in vitro product, AFG1-lysine, was characterized by UV, fluorescence, 1H- and 13C-NMR spectroscopy and fast atom bombardment MS. A competitive enzyme-linked immunoassay for this adduct was established using polyclonal antibodies to AFB1 and this was used together with an HPLC-fluorescence technique to quantitate the in vivo formation of AFG1-albumin adducts in comparison to AFB1.	Lysine	27-33	AFG1 like ATPase	Homo sapiens	343-347
1899057-3	1991	The in vitro product, AFG1-lysine, was characterized by UV, fluorescence, 1H- and 13C-NMR spectroscopy and fast atom bombardment MS. A competitive enzyme-linked immunoassay for this adduct was established using polyclonal antibodies to AFB1 and this was used together with an HPLC-fluorescence technique to quantitate the in vivo formation of AFG1-albumin adducts in comparison to AFB1.	Hydrogen	74-76	AFG1 like ATPase	Homo sapiens	22-26
1899057-3	1991	The in vitro product, AFG1-lysine, was characterized by UV, fluorescence, 1H- and 13C-NMR spectroscopy and fast atom bombardment MS. A competitive enzyme-linked immunoassay for this adduct was established using polyclonal antibodies to AFB1 and this was used together with an HPLC-fluorescence technique to quantitate the in vivo formation of AFG1-albumin adducts in comparison to AFB1.	13c	82-85	AFG1 like ATPase	Homo sapiens	22-26
1899057-5	1991	The levels of AFG1-albumin adducts were determined to be 5.7- and 2.8-fold lower than with equivalent doses of AFB1 as determined by immunoassay and HPLC fluorescence respectively.	Aflatoxin B1	111-115	AFG1 like ATPase	Homo sapiens	14-18
7532124-1	1994	To find out if the presumed intake of dietary aflatoxins (AFB1 and AFG1) has adverse effect on the liver of Ghanaians, the toxins were measured in serum, urine and faecal specimens obtained from a group of apparently healthy Ghanaian adults.	Aflatoxins	46-56	AFG1 like ATPase	Homo sapiens	67-71