PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
20626654-6	2010	Overexpression of SIG6 rescued the chlorophyll deficiency in dg1 cotyledons but not in young leaves.	Chlorophyll	35-46	RNApolymerase sigma-subunit F	Arabidopsis thaliana	18-22
25505479-5	2014	We made efforts to reduce the complexity by genetic crosses of Arabidopsis single mutants (with focus on a chlorophyll-deficient sig6 line) to generate double knockout lines.	Chlorophyll	107-118	RNApolymerase sigma-subunit F	Arabidopsis thaliana	129-133
22527751-7	2012	Our data show that SIG3 and SIG6 also participate in transcription initiation of the large and the small atp operon, respectively.	Adenosine Triphosphate	105-108	RNApolymerase sigma-subunit F	Arabidopsis thaliana	28-32
22527751-8	2012	We propose that SIG2 might be the factor responsible for coordinating the basal transcription of the plastid atp genes and that SIG3 and SIG6 might serve to modulate plastid atp expression with respect to physiological and environmental conditions.	Adenosine Triphosphate	174-177	RNApolymerase sigma-subunit F	Arabidopsis thaliana	137-141
22527751-9	2012	However, we observe that in the sigma mutants (sig2, sig3 and sig6) the deficiency in the recognition of specific atp promoters is largely balanced by mRNA stabilization and/or by activation of otherwise silent promoters, indicating that the rate-limiting step for expression of the atp operons is mostly post-transcriptional.	Adenosine Triphosphate	114-117	RNApolymerase sigma-subunit F	Arabidopsis thaliana	62-66
22527751-9	2012	However, we observe that in the sigma mutants (sig2, sig3 and sig6) the deficiency in the recognition of specific atp promoters is largely balanced by mRNA stabilization and/or by activation of otherwise silent promoters, indicating that the rate-limiting step for expression of the atp operons is mostly post-transcriptional.	Adenosine Triphosphate	283-286	RNApolymerase sigma-subunit F	Arabidopsis thaliana	62-66
19273469-0	2009	Characterization of soldat8, a suppressor of singlet oxygen-induced cell death in Arabidopsis seedlings.	Singlet Oxygen	45-59	RNApolymerase sigma-subunit F	Arabidopsis thaliana	20-27
19273469-12	2009	Suppression of (1)O2-mediated cell death in young flu/soldat8 seedlings seems to be due to a transiently enhanced acclimation at the beginning of seedling development caused by the initial disturbance of plastid homeostasis.	Singlet Oxygen	18-20	RNApolymerase sigma-subunit F	Arabidopsis thaliana	54-61
32002990-9	2020	RESULTS: Among six sig mutants, only the sig6 mutant significantly accumulated chlorophyll after FR BOG treatment, similar to the phyA mutant.	Chlorophyll	79-90	RNApolymerase sigma-subunit F	Arabidopsis thaliana	41-45
32002990-10	2020	SIG6 appears to control protochlorophyllide accumulation by contributing to the regulation of tetrapyrrole biosynthesis associated with glutamyl-tRNA reductase (HEMA1) function, select phytochrome-interacting factor genes (PIF4 and PIF6), and PENTA1, which regulates PORA mRNA translation after FR exposure.	Protochlorophyllide	24-43	RNApolymerase sigma-subunit F	Arabidopsis thaliana	0-4
32002990-10	2020	SIG6 appears to control protochlorophyllide accumulation by contributing to the regulation of tetrapyrrole biosynthesis associated with glutamyl-tRNA reductase (HEMA1) function, select phytochrome-interacting factor genes (PIF4 and PIF6), and PENTA1, which regulates PORA mRNA translation after FR exposure.	Tetrapyrroles	94-106	RNApolymerase sigma-subunit F	Arabidopsis thaliana	0-4
30943245-5	2019	This effect can be observed on a genome-wide scale, as the loss of the plastid sigma transcription factor SIG6 prevents DNA rearrangements by favoring conservative repair in the presence of ciprofloxacin-induced DNA damage or in the absence of plastid genome maintenance actors such as WHY1/WHY3, RECA1 and POLIB.	Ciprofloxacin	190-203	RNApolymerase sigma-subunit F	Arabidopsis thaliana	106-110