PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
31740007-2	2020	In this study, a xerogel (RFX) prepared by an optimized conventional sol-gel method and a lignin-based activated carbon (KLP) obtained via chemical activation were used in batch and fixed-bed adsorption experiments.	Carbon	113-119	kinesin family member 1B	Homo sapiens	121-124
32506913-5	2020	Preclinical ADME and in vivo target engagement studies of 13B support further development of 6-phenylhexanamide derivatives as therapeutic agents for human CMT2A.	CHEMBL3741121	58-61	kinesin family member 1B	Homo sapiens	156-161
32506913-5	2020	Preclinical ADME and in vivo target engagement studies of 13B support further development of 6-phenylhexanamide derivatives as therapeutic agents for human CMT2A.	6-phenylhexanamide	93-111	kinesin family member 1B	Homo sapiens	156-161
33074106-2	2020	Here, pharmacological activation of endogenous normal mitofusins overcame dominant inhibitory effects of CMT2A mutants in reprogrammed human patient motor neurons, reversing hallmark mitochondrial stasis and fragmentation independent of causal MFN2 mutation.	mitofusins	54-64	kinesin family member 1B	Homo sapiens	105-110
32310125-5	2020	The present work aims to study the adsorption of BPA from aqueous solutions onto carbonaceous materials, e.g., a synthesized carbon xerogel (RFX), a chemical-activated carbon from Kraft lignin (KLP) and a commercial activated carbon (F400) for comparative purposes.	bisphenol A	49-52	kinesin family member 1B	Homo sapiens	194-197
32733278-1	2020	A causal relationship between Mitofusin (MFN) 2 gene mutations and the hereditary axonal neuropathy Charcot-Marie-Tooth disease type 2A (CMT2A) was described over 15 years ago.	mitofusin	30-39	kinesin family member 1B	Homo sapiens	137-142
31740007-4	2020	RFX and KLP materials reached the equilibrium adsorption in only 24 h, whereas F400 activated carbon required 48 h. In addition, F400 and KLP adsorbents showed higher equilibrium adsorption capacity values (qe = 0.40 and 0.22 kg/kg, for F400 and KLP, respectively) than that obtained for the xerogel (qe = 0.08 kg/kg).	Carbon	94-100	kinesin family member 1B	Homo sapiens	138-141
31740007-4	2020	RFX and KLP materials reached the equilibrium adsorption in only 24 h, whereas F400 activated carbon required 48 h. In addition, F400 and KLP adsorbents showed higher equilibrium adsorption capacity values (qe = 0.40 and 0.22 kg/kg, for F400 and KLP, respectively) than that obtained for the xerogel (qe = 0.08 kg/kg).	Carbon	94-100	kinesin family member 1B	Homo sapiens	138-141
23165935-7	2013	Injection of hyperpolarized [1-(13)C]lactate with unlabeled sodium pyruvate in the dissolution buffer, provided exchange rate constants Klp for kidney and vascular regions of interest.	[1-(13)c]lactate	28-44	kinesin family member 1B	Homo sapiens	136-139
31664033-8	2019	CMT2A-related mutations, mapping to different functional zones of MFN2, lead to changes in GTP hydrolysis and homo/hetero-association ability.	Guanosine Triphosphate	91-94	kinesin family member 1B	Homo sapiens	0-5
27122668-12	2016	CONCLUSION: The gene-environment interaction between the KIF1B rs17401966 variant and alcohol consumption may contribute to the development of HCC in Chinese individuals.	Alcohols	86-93	kinesin family member 1B	Homo sapiens	57-62
23165935-7	2013	Injection of hyperpolarized [1-(13)C]lactate with unlabeled sodium pyruvate in the dissolution buffer, provided exchange rate constants Klp for kidney and vascular regions of interest.	SODIUM PYRUVATE	60-75	kinesin family member 1B	Homo sapiens	136-139
33818493-0	2021	Mitofusin activation enhances mitochondrial motility and promotes neuroregeneration in CMT2A.	mitofusin	0-9	kinesin family member 1B	Homo sapiens	87-92
23427948-1	2012	OBJECTIVE: Acetylsalicylic acid (ASA) and clopidogrel (KLP) therapy is associated with the high degree of variability in response to the drug and some patients are drug-resistant.	Clopidogrel	42-53	kinesin family member 1B	Homo sapiens	55-58
23427948-5	2012	Good response to KLP treatment with 20 micromol/l ADP-induced platelet aggregation inhibition < 60% reached 66.1% of patients, partial response with 60-70% inhibition 13.9% of patients and poor response > 70% in 20% of patients.	Adenosine Diphosphate	50-53	kinesin family member 1B	Homo sapiens	17-20
19812251-0	2009	A mutation associated with CMT2A neuropathy causes defects in Fzo1 GTP hydrolysis, ubiquitylation, and protein turnover.	Guanosine Triphosphate	67-70	kinesin family member 1B	Homo sapiens	27-32
19812251-5	2009	One mutation (analogous to the CMT2A I213T substitution in the GTPase domain of Mfn2) not only abolishes GTP hydrolysis and mitochondrial membrane fusion but also reduces Mdm30-mediated ubiquitylation and degradation of the mutant protein.	Guanosine Triphosphate	63-66	kinesin family member 1B	Homo sapiens	31-36
19812251-7	2009	These studies identify diverse and unexpected effects of CMT2A mutations, including a possible role for mitofusin ubiquitylation and degradation in CMT2A pathogenesis, and provide evidence for a novel link between Fzo1 GTP hydrolysis, ubiquitylation, and mitochondrial fusion.	Guanosine Triphosphate	219-222	kinesin family member 1B	Homo sapiens	57-62
18726616-7	2008	Furthermore, KIF1B beta-mutant tumors are uniquely enriched for pathways related to glutamate metabolism and the oxidative stress response.	Glutamic Acid	84-93	kinesin family member 1B	Homo sapiens	13-18