PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
33294259-0	2020	RBMX contributes to hepatocellular carcinoma progression and sorafenib resistance by specifically binding and stabilizing BLACAT1.	Sorafenib	61-70	RNA binding motif protein X-linked	Homo sapiens	0-4
33294259-4	2020	In this study, we have identified RBMX significantly contributes to the tumorigenesis and sorafenib resistance of hepatocellular carcinoma (HCC).	Sorafenib	90-99	RNA binding motif protein X-linked	Homo sapiens	34-38
33294259-6	2020	The HCC cell"s viability, proliferation, and sorafenib resistance ability were both increased when RBMX was overexpressed.	Sorafenib	45-54	RNA binding motif protein X-linked	Homo sapiens	99-103
33294259-8	2020	Further, we found that the autophagy level was increased in HCC cells, which RBMX was up regulated, with sorafenib processing.	Sorafenib	105-114	RNA binding motif protein X-linked	Homo sapiens	77-81
34638274-5	2021	Low RBMX expression was associated with better response to anthracycline-containing first-line treatment.	Anthracyclines	59-72	RNA binding motif protein X-linked	Homo sapiens	4-8
32317327-6	2020	Mechanistically, RBMX binds to HIV-1 proviral DNA at the LTR downstream region and maintains the repressive trimethylation of histone H3 lysine 9 (H3K9me3), leading to a blockage of the recruitment of the positive transcription factor phosphorylated RNA polymerase II (RNA pol II) and consequential impediment of transcription elongation.	Lysine	137-143	RNA binding motif protein X-linked	Homo sapiens	17-21
29156680-10	2017	We find that vemurafenib-resistant cells show amplification of chromosome 5 and de novo mutations in the RBM (RNA-binding motifs) genes family (i.e. RBMX, RBM10).	Vemurafenib	13-24	RNA binding motif protein X-linked	Homo sapiens	149-153