PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
17272267-9	2007	In two independent ICAT experiments, we identified 427 cysteine-containing striatal proteins among which approximately 66% (203 proteins) were detected in both ICAT experiments.	Cysteine	55-63	catenin beta interacting protein 1	Mus musculus	19-23
18649742-1	2008	OBJECTIVE: To investigate the effect of 17 beta-estradiol (E2) on T-type calcium currents (ICaT) in spermatogenic cells.	Estradiol	40-57	catenin beta interacting protein 1	Mus musculus	91-95
18649742-1	2008	OBJECTIVE: To investigate the effect of 17 beta-estradiol (E2) on T-type calcium currents (ICaT) in spermatogenic cells.	Calcium	73-80	catenin beta interacting protein 1	Mus musculus	91-95
17272267-9	2007	In two independent ICAT experiments, we identified 427 cysteine-containing striatal proteins among which approximately 66% (203 proteins) were detected in both ICAT experiments.	Cysteine	55-63	catenin beta interacting protein 1	Mus musculus	160-164
16322052-6	2006	An examination of ionic conductances generated by firing revealed that calcium entry through ICAT controls the emergence of the mAChR-mediated ADP.	Calcium	71-78	catenin beta interacting protein 1	Mus musculus	93-97
16894933-1	2006	OBJECTIVE: To investigate the effects of Flunarizine (Flu) on T-type calcium currents (ICaT) in spermatogenic cells.	Flunarizine	41-52	catenin beta interacting protein 1	Mus musculus	87-91
16894933-1	2006	OBJECTIVE: To investigate the effects of Flunarizine (Flu) on T-type calcium currents (ICaT) in spermatogenic cells.	Calcium	69-76	catenin beta interacting protein 1	Mus musculus	87-91
17028301-5	2007	We applied a quantitative proteomics analysis technique with the cleavable ICAT reagent to quantitate relative changes in protein levels of the hippocampal PSD in response to morphine administration.	Morphine	175-183	catenin beta interacting protein 1	Mus musculus	75-79
16894933-3	2006	RESULTS: Flu of 0.1, 1, 10, 100 micromol/L inhibited ICaT in mouse spermatogenic cells significantly with the K50 value of 0.289 micromol/L (P < 0.05).	Flunarizine	9-12	catenin beta interacting protein 1	Mus musculus	53-57
16894933-5	2006	CONCLUSION: Flu has significant inhibitory effects on ICaT in mouse spermatogenic cells, with concentration dependence.	Flunarizine	12-15	catenin beta interacting protein 1	Mus musculus	54-58
16322052-6	2006	An examination of ionic conductances generated by firing revealed that calcium entry through ICAT controls the emergence of the mAChR-mediated ADP.	Adenosine Diphosphate	143-146	catenin beta interacting protein 1	Mus musculus	93-97
34118593-6	2021	A dual-luciferase reporter assay was used to confirm the interactions between CTNNBIP1 and miR-486-3p.	mir-486-3p	91-101	catenin beta interacting protein 1	Mus musculus	78-86
16216825-5	2006	The results presented here show that labeling of proteins with cleavable ICAT is possible and may even be improved in strong denaturing buffers containing both SDS at a concentration higher than 0.5% (w/v) and urea.	Sodium Dodecyl Sulfate	160-163	catenin beta interacting protein 1	Mus musculus	73-77
16216825-5	2006	The results presented here show that labeling of proteins with cleavable ICAT is possible and may even be improved in strong denaturing buffers containing both SDS at a concentration higher than 0.5% (w/v) and urea.	Urea	210-214	catenin beta interacting protein 1	Mus musculus	73-77
16216825-6	2006	These results open the possibility of applying the ICAT strategy to complex samples containing very hydrophobic proteins solubilized in urea-SDS buffers.	Urea	136-140	catenin beta interacting protein 1	Mus musculus	51-55
16216825-6	2006	These results open the possibility of applying the ICAT strategy to complex samples containing very hydrophobic proteins solubilized in urea-SDS buffers.	Sodium Dodecyl Sulfate	141-144	catenin beta interacting protein 1	Mus musculus	51-55
9751151-9	1998	The results suggest that neurons activate, via diffusible and degradable factors, a subset of Schwann cell cAMP pathways leading to expression of IcaT, and activate additional non-cAMP pathways that lead to expression of 04.	Cyclic AMP	107-111	catenin beta interacting protein 1	Mus musculus	146-150
34118593-11	2021	CTNNBIP1 was confirmed as a target of miR-486-3p.	mir-486-3p	38-48	catenin beta interacting protein 1	Mus musculus	0-8
34118593-14	2021	CONCLUSION: This study demonstrated that miR-486-3p targets CTNNBIP1, thus activating the Wnt/beta-catenin signaling pathway to promote osteogenesis of BMSCs.	mir-486	41-48	catenin beta interacting protein 1	Mus musculus	60-68
25321480-6	2014	Treatment with a miR-29b reduced the reporter activity of a luciferase-ICAT 3"-UTR construct whereas a control (scrambled) miRNA oligonucleotide did not, indicating that miR-29b directly targets the 3"-UTR of ICAT.	Oligonucleotides	129-144	catenin beta interacting protein 1	Mus musculus	71-75
32531088-7	2021	Within IMQ-induced psoriasis-like dermatitis in mice, CTNNBIP1 silence further aggravated psoriatic phenotypes.	Imiquimod	7-10	catenin beta interacting protein 1	Mus musculus	54-62
33390980-0	2020	Suppression of mICAT in Mouse Small Intestinal Myocytes by General Anaesthetic Ketamine and its Recovery by TRPC4 Agonist (-)-englerin A.	Ketamine	79-87	catenin beta interacting protein 1	Mus musculus	15-20
33390980-0	2020	Suppression of mICAT in Mouse Small Intestinal Myocytes by General Anaesthetic Ketamine and its Recovery by TRPC4 Agonist (-)-englerin A.	englerin	126-134	catenin beta interacting protein 1	Mus musculus	15-20
33390980-3	2020	Here we aimed to characterize the effects of one of the most commonly used "dissociative anesthetics", ketamine, on mICAT.	Ketamine	103-111	catenin beta interacting protein 1	Mus musculus	116-121
33390980-4	2020	Patch-clamp and tensiometry techniques were used to investigate the mechanisms of the inhibitory effects of ketamine on mICAT in single mouse ileal myocytes, as well as on intestinal motility.	Ketamine	108-116	catenin beta interacting protein 1	Mus musculus	120-125
33390980-5	2020	Ketamine (100 microM) strongly inhibited both carbachol- and GTPgammaS-induced mICAT.	Ketamine	0-8	catenin beta interacting protein 1	Mus musculus	79-84
33390980-5	2020	Ketamine (100 microM) strongly inhibited both carbachol- and GTPgammaS-induced mICAT.	Carbachol	46-55	catenin beta interacting protein 1	Mus musculus	79-84
33390980-5	2020	Ketamine (100 microM) strongly inhibited both carbachol- and GTPgammaS-induced mICAT.	Guanosine 5'-O-(3-Thiotriphosphate)	61-70	catenin beta interacting protein 1	Mus musculus	79-84
33390980-9	2020	Importantly, inhibited by ketamine mICAT could be restored by direct TRPC4 agonist (-)-englerin A.	Ketamine	26-34	catenin beta interacting protein 1	Mus musculus	35-40
33390980-10	2020	We identified mICAT as a novel target for ketamine.	Ketamine	42-50	catenin beta interacting protein 1	Mus musculus	14-19
31872561-5	2019	In the presence of RyR mutations, frequent spontaneous calcium leaks from sarcoplasmic reticulum (SR) initiated calcium waves, which upon reaching the cell periphery produced delayed afterdepolarizations (DADs) via sodium-calcium exchanger (NCX) and T-type calcium (ICaT ) channel activation.	Calcium	55-62	catenin beta interacting protein 1	Mus musculus	266-270
31872561-5	2019	In the presence of RyR mutations, frequent spontaneous calcium leaks from sarcoplasmic reticulum (SR) initiated calcium waves, which upon reaching the cell periphery produced delayed afterdepolarizations (DADs) via sodium-calcium exchanger (NCX) and T-type calcium (ICaT ) channel activation.	Calcium	112-119	catenin beta interacting protein 1	Mus musculus	266-270
31872561-5	2019	In the presence of RyR mutations, frequent spontaneous calcium leaks from sarcoplasmic reticulum (SR) initiated calcium waves, which upon reaching the cell periphery produced delayed afterdepolarizations (DADs) via sodium-calcium exchanger (NCX) and T-type calcium (ICaT ) channel activation.	Sodium	215-221	catenin beta interacting protein 1	Mus musculus	266-270
31872561-5	2019	In the presence of RyR mutations, frequent spontaneous calcium leaks from sarcoplasmic reticulum (SR) initiated calcium waves, which upon reaching the cell periphery produced delayed afterdepolarizations (DADs) via sodium-calcium exchanger (NCX) and T-type calcium (ICaT ) channel activation.	Calcium	112-119	catenin beta interacting protein 1	Mus musculus	266-270
31872561-5	2019	In the presence of RyR mutations, frequent spontaneous calcium leaks from sarcoplasmic reticulum (SR) initiated calcium waves, which upon reaching the cell periphery produced delayed afterdepolarizations (DADs) via sodium-calcium exchanger (NCX) and T-type calcium (ICaT ) channel activation.	Calcium	112-119	catenin beta interacting protein 1	Mus musculus	266-270
27829235-6	2016	In the mechanism study, CARF was found to interact with beta-catenin, impaired the interaction between beta-catenin and ICAT, and activated beta-catenin/TCF signaling.	carf	24-28	catenin beta interacting protein 1	Mus musculus	120-124
25321480-6	2014	Treatment with a miR-29b reduced the reporter activity of a luciferase-ICAT 3"-UTR construct whereas a control (scrambled) miRNA oligonucleotide did not, indicating that miR-29b directly targets the 3"-UTR of ICAT.	Oligonucleotides	129-144	catenin beta interacting protein 1	Mus musculus	209-213
25175916-0	2014	MiR-424-5p reversed epithelial-mesenchymal transition of anchorage-independent HCC cells by directly targeting ICAT and suppressed HCC progression.	mir-424-5p	0-10	catenin beta interacting protein 1	Mus musculus	111-115
25175916-6	2014	Further study demonstrated that miR-424-5p reversed resistance to anoikis and EMT of HCCs by directly targeting ICAT and further maintaining the E-cadherin/beta-catanin complex on the cellular membrance.	mir-424-5p	32-42	catenin beta interacting protein 1	Mus musculus	112-116