PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
27434365-0	2016	Tp-Te interval predicts heart rate reduction after fingolimod administration in patients with multiple sclerosis.	Fingolimod Hydrochloride	51-61	transmembrane phosphatase with tensin homology	Homo sapiens	0-5
28912075-14	2018	The ratio of Tp-Te to QTc was also significantly increased after pulse steroid therapy (0,18+-0,03 vs 0,20+-0,03 p:0,009).	qtc	22-25	transmembrane phosphatase with tensin homology	Homo sapiens	13-18
28912075-14	2018	The ratio of Tp-Te to QTc was also significantly increased after pulse steroid therapy (0,18+-0,03 vs 0,20+-0,03 p:0,009).	Steroids	71-78	transmembrane phosphatase with tensin homology	Homo sapiens	13-18
27434365-12	2016	CONCLUSIONS: This study firstly demonstrates that prolonged Tp-Te interval may identify those MS patients treated with fingolimod at higher risk of having significant, asymptomatic HR reduction during clinical observation.	Fingolimod Hydrochloride	119-129	transmembrane phosphatase with tensin homology	Homo sapiens	60-65
24964631-6	2014	Tp-Te interval prolonged with increasing of water As (r = 0.199, P = 0.023), as well prolonged with increasing of ERbeta mRNA expression (r = 0.205, P = 0.019).	Water	44-49	transmembrane phosphatase with tensin homology	Homo sapiens	0-5
26350112-7	2015	TPTE sero-positivity was not associated with tumor stage, tumor histology, gender or age.	sero	5-9	transmembrane phosphatase with tensin homology	Homo sapiens	0-4
26350112-8	2015	Multivariate analysis indicated that TPTE sero-positivity is associated with prolonged survival in patients with lung cancer, but established prognostic factors for survival prediction such as stage and histology remain indispensable.	sero	42-46	transmembrane phosphatase with tensin homology	Homo sapiens	37-41
26350112-10	2015	TPTE sero-reactivity has moderate sensitivity and specificity for diagnosing lung cancer and is a positive prognostic marker.	sero	5-9	transmembrane phosphatase with tensin homology	Homo sapiens	0-4
24964631-8	2014	CONCLUSION: chronic arsenic exposure can potentially disturb ERbeta mRNA expression, as well as there are possible relationship between ERbeta mRNA abnormal expression and Q-Tc interval prolongation and Tp-Te interval prolonged caused by arsenic.	Arsenic	238-245	transmembrane phosphatase with tensin homology	Homo sapiens	203-208
12972125-1	2003	Comparing patients treated after myocardial infarction with amiodarone or with placebo, we found a significant rate-dependent prolongation of TpTe interval in patients who received amiodarone.	Amiodarone	60-70	transmembrane phosphatase with tensin homology	Homo sapiens	142-146
20085555-0	2010	The effect of sertindole on QTD and TPTE.	sertindole	14-24	transmembrane phosphatase with tensin homology	Homo sapiens	36-40
12972125-1	2003	Comparing patients treated after myocardial infarction with amiodarone or with placebo, we found a significant rate-dependent prolongation of TpTe interval in patients who received amiodarone.	Amiodarone	181-191	transmembrane phosphatase with tensin homology	Homo sapiens	142-146
12972125-3	2003	Assuming that TpTe reflects transmural repolarization heterogeneity, our findings suggest that heterogeneity and arrhythmic risk are increased by amiodarone.	Amiodarone	146-156	transmembrane phosphatase with tensin homology	Homo sapiens	14-18
33064338-10	2021	In a univariate regression analysis, body mass index, smoking status, Tp-Te, and Tp-Te/QTc were significantly associated with the severity of AHI in OSA.	qtc	87-90	transmembrane phosphatase with tensin homology	Homo sapiens	81-86