PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
33853611-9	2021	Mechanistically, miR-340-5p was validated to be targeted by LINC00641 and knockdown of miR-340-5p counteracted LINC00641 silencing-mediated inhibition of RCC progression.	mir-340-5p	17-27	long intergenic non-protein coding RNA 641	Homo sapiens	60-69
33853611-9	2021	Mechanistically, miR-340-5p was validated to be targeted by LINC00641 and knockdown of miR-340-5p counteracted LINC00641 silencing-mediated inhibition of RCC progression.	mir-340-5p	87-97	long intergenic non-protein coding RNA 641	Homo sapiens	111-120
33853611-11	2021	CONCLUSIONS: These results suggested that LINC00641 promoted the progression of RCC by sponging miR-340-5p.	mir-340-5p	96-106	long intergenic non-protein coding RNA 641	Homo sapiens	42-51
31572456-8	2019	Four lncRNAs (FLJ30679, LINC01193, LINC00692, and LINC00641) were observed to be up-regulated in DFU.	5,5-dimethyl-3-(3-fluorophenyl)-4-(4-methylsulfonyl)phenyl-2(5H)-furanone	97-100	long intergenic non-protein coding RNA 641	Homo sapiens	50-59
31490021-10	2020	miR-194-5p overexpression reversed the effects of LINC00641 on cell proliferation, cycle, apoptosis, migration, as well as invasion.	mir-194-5p	0-10	long intergenic non-protein coding RNA 641	Homo sapiens	50-59
32917936-0	2020	LINC00641/miR-582-5p mediate oxaliplatin resistance by activating autophagy in gastric adenocarcinoma.	Oxaliplatin	29-40	long intergenic non-protein coding RNA 641	Homo sapiens	0-9
32917936-3	2020	We aim to find the roles that LINC00641 and miR-582-5p play in regulating oxaliplatin resistance.	Oxaliplatin	74-85	long intergenic non-protein coding RNA 641	Homo sapiens	30-39
32917936-6	2020	LINC00641 expression was associated with prognosis and oxaliplatin resistance in patients with gastric adenocarcinoma.	Oxaliplatin	55-66	long intergenic non-protein coding RNA 641	Homo sapiens	0-9
32917936-8	2020	Moreover, LINC00641 was highly expressed in oxaliplatin-resistant cell lines and miR-582-5p was down-regulated.	Oxaliplatin	44-55	long intergenic non-protein coding RNA 641	Homo sapiens	10-19
32917936-11	2020	Further experiments indicated that down-regulation of LINC00641 inhibited the autophagy process, making gastric cancer cells more sensitive to oxaliplatin.	Oxaliplatin	143-154	long intergenic non-protein coding RNA 641	Homo sapiens	54-63
32917936-12	2020	LINC00641 and miR-582-5p are biomarkers for predicting overall survival, as they were involved in regulating oxaliplatin resistance by altering autophagy in gastric adenocarcinoma.	Oxaliplatin	109-120	long intergenic non-protein coding RNA 641	Homo sapiens	0-9
32714145-2	2020	This study aims to investigate the effects of long non-coding RNA LINC00641 on the ketamine-induced neural injury.	Ketamine	83-91	long intergenic non-protein coding RNA 641	Homo sapiens	66-75
32714145-4	2020	Ketamine-induced aberrant expression levels of LINC00641, miR-497-5p and brain-derived neurotrophic factor (BDNF) were examined by qRT-PCR.	Ketamine	0-8	long intergenic non-protein coding RNA 641	Homo sapiens	47-56
32714145-8	2020	Results: Ketamine induced the apoptosis of PC12 cells, accompanied by down-regulation of LINC00641 and BDNF, and up-regulation of miR-497-5p.	Ketamine	9-17	long intergenic non-protein coding RNA 641	Homo sapiens	89-98
32714145-11	2020	Conclusion: Collectively, LINC00641/miR-497-5p/BDNF axis was validated to be an important signaling pathway in modulating ketamine-induced neural injury.	Ketamine	122-130	long intergenic non-protein coding RNA 641	Homo sapiens	26-35