PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
17983375-10	2007	Finally, serum IL-5 and Bronchoalveolar lavage fluid eotaxin-2 levels were abolished in allergen exposed CD4-/- mice to levels seen in saline exposed WT mice.	Sodium Chloride	135-141	chemokine (C-C motif) ligand 24	Mus musculus	53-62
14610483-6	2003	RESULTS: Eotaxin-2 and IL-5 cooperatively promoted eosinophil accumulation, IL-13 production, and AHR to methacholine.	Methacholine Chloride	105-117	chemokine (C-C motif) ligand 24	Mus musculus	9-18
32039405-10	2020	Blocking CCL24 using CM-101 robustly reduced liver damage in 3 experimental animal models (MCD, STAM and TAA), as demonstrated by attenuation of liver fibrosis and NAFLD activity score.	Thioacetamide	105-108	chemokine (C-C motif) ligand 24	Mus musculus	9-14
33463104-12	2020	Qur-40 could show an enhancement effect on DEC for the reduction of IL-4, IL-5, IgE, EPO, and eotaxin 2.	qur-40	0-6	chemokine (C-C motif) ligand 24	Mus musculus	94-103
34221188-7	2021	Western blot analysis showed that expression of CCL24 was significantly increased under high-glucose conditions.	Glucose	93-100	chemokine (C-C motif) ligand 24	Mus musculus	48-53
34221188-8	2021	Stimulation with high glucose (35 mmol/L) resulted in an increase in CCL24 expression in the first 48 hours but changed little after 72 hours.	Glucose	22-29	chemokine (C-C motif) ligand 24	Mus musculus	69-74
34221188-12	2021	Compared with the control group, the podocyte inflammatory response induced by high glucose after CCL24 knockout was significantly increased.	Glucose	84-91	chemokine (C-C motif) ligand 24	Mus musculus	98-103
33792561-7	2021	However, UDP-G increased chemokinesis in eosinophils and enhanced their response to the eosinophil chemoattractant, CCL24.	Uridine Diphosphate Glucose	9-14	chemokine (C-C motif) ligand 24	Mus musculus	116-121
32524776-5	2020	Interestingly, we observed distinct large bright CD11b positive cells in the bronchoalveolar lavage, upregulation of lung vascular and alveolar ATP synthase as well as plasminogen and bronchiolar angiostatin expression in ozone-exposed mice, platelet and neutrophil accumulation in the lung vasculature and an eotaxin-2, IL-16, CXCL5, CXCL12, and CXCL13 dominant inflammatory response leading to lung injury.	Ozone	222-227	chemokine (C-C motif) ligand 24	Mus musculus	310-319
31019244-5	2019	The inhibitory action of glucagon occurred in parallel with reduction of OVA-induced generation of IL-4, IL-5, IL-13, TNF-alpha, eotaxin-1/CCL11, and eotaxin-2/CCL24 but not MDC/CCL22 and TARC/CCL17.	Glucagon	25-33	chemokine (C-C motif) ligand 24	Mus musculus	150-159
31611798-7	2019	Additionally, t-TUCB markedly inhibited eotaxin-2, an eosinophil-specific chemokine, which was only marginally reduced by PTUPB and remained elevated in celecoxib-treated mice.	4-(4-(3-(4-trifluoromethoxy-phenyl)ureido)cyclohexyloxy)benzoic acid	14-20	chemokine (C-C motif) ligand 24	Mus musculus	40-49
31611798-7	2019	Additionally, t-TUCB markedly inhibited eotaxin-2, an eosinophil-specific chemokine, which was only marginally reduced by PTUPB and remained elevated in celecoxib-treated mice.	celecoxib	153-162	chemokine (C-C motif) ligand 24	Mus musculus	40-49
31019244-5	2019	The inhibitory action of glucagon occurred in parallel with reduction of OVA-induced generation of IL-4, IL-5, IL-13, TNF-alpha, eotaxin-1/CCL11, and eotaxin-2/CCL24 but not MDC/CCL22 and TARC/CCL17.	Glucagon	25-33	chemokine (C-C motif) ligand 24	Mus musculus	160-165
29910732-8	2018	Administration of BLM and DNCB increased the levels of IL-4 and IL-13 production in spleen cells and eotaxin-2 mRNA expression in dorsal skin, compared to NC/Nga mice treated with DNCB alone.	Dinitrochlorobenzene	26-30	chemokine (C-C motif) ligand 24	Mus musculus	101-110
28785009-6	2017	Moreover, alveolar macrophages performed significant enhancement of M2 functions, especially CCL24 secretion, in the Abt mice challenged with B16/F10 melanoma.	abt	117-120	chemokine (C-C motif) ligand 24	Mus musculus	93-98
29225189-6	2018	Luteolin and cyclosporine A (CsA) which was a positive control also substantially reduced OVA-specific IgE levels, eotaxin 2 levels, and CCR3 expression in BAL Fluid.	Cyclosporine	13-27	chemokine (C-C motif) ligand 24	Mus musculus	115-124
29225189-6	2018	Luteolin and cyclosporine A (CsA) which was a positive control also substantially reduced OVA-specific IgE levels, eotaxin 2 levels, and CCR3 expression in BAL Fluid.	Cyclosporine	29-32	chemokine (C-C motif) ligand 24	Mus musculus	115-124
29782549-7	2018	Bleomycin administration stimulated the mRNA expression of the profibrotic cytokines IL-13 and IL-4 and the M2 markers including arginase 1, Fizz1/Retnla, Ccl17 and Ccl24 in cells collected from broncho-alveolar lavage fluids (BALF), and S1pr2 deletion markedly diminished the stimulated expression of these genes.	Bleomycin	0-9	chemokine (C-C motif) ligand 24	Mus musculus	165-170
28785009-7	2017	Adoptive transfer of normal alveolar macrophages or antibody neutralization of CCL24 significantly recovered the decrease of gammadeltaT17 cells and rescued the defect anti-tumor response of Abt mice, indicating the elevated amount of alveolar macrophage-derived CCL24 inhibited gammadeltaT cell mediated anti-tumor response.	abt	191-194	chemokine (C-C motif) ligand 24	Mus musculus	79-84
28078007-10	2016	Eotaxin-2 administration led to a significant elevation of high cholesterol diet-induced atherosclerosis, and of TLR4 expression in B6.129S7-Ldlrtm1Her /J but not Ldlr-/--/-/Tlr4-/- mice.	Cholesterol	64-75	chemokine (C-C motif) ligand 24	Mus musculus	0-9
23822876-7	2013	The inhibition of the cysteinyl LT (CysLT) signaling pathway by montelukast inhibited IL-4, IL-5, eotaxin-2, and regulated upon activation normal T cell expressed and presumably secreted expression in the developing lungs of Foxa2 gene-targeted mice.	montelukast	64-75	chemokine (C-C motif) ligand 24	Mus musculus	98-107
27165276-6	2016	Additionally, in vivo and vitro experiments indicated that AQP3 induced the production of some chemokines such as CCL24 and CCL22 through regulating the amount of cellular H2O2 in M2 polarized alveolar macrophages.	Hydrogen Peroxide	172-176	chemokine (C-C motif) ligand 24	Mus musculus	114-119
25267883-7	2015	Enhanced CCL24 expression was also observed in GM-CSF-induced BMDMs and zymosan-elicited, thioglycolate-elicited and naive peritoneal macrophages.	Zymosan	72-79	chemokine (C-C motif) ligand 24	Mus musculus	9-14
25267883-7	2015	Enhanced CCL24 expression was also observed in GM-CSF-induced BMDMs and zymosan-elicited, thioglycolate-elicited and naive peritoneal macrophages.	Thioglycolates	90-103	chemokine (C-C motif) ligand 24	Mus musculus	9-14
27071418-7	2016	TSA treatment significantly decreased protein expression of IL-5, IL-13, CCL11 and CCL24 in the lung homogenates from Alternaria extract-challenged mice or rmIL-33-challenged mice compared with vehicle treatment (p<0.05).	trichostatin A	0-3	chemokine (C-C motif) ligand 24	Mus musculus	83-88
26481537-0	2015	Chelidonine, a principal isoquinoline alkaloid of Chelidonium majus, attenuates eosinophilic airway inflammation by suppressing IL-4 and eotaxin-2 expression in asthmatic mice.	chelidonine	0-11	chemokine (C-C motif) ligand 24	Mus musculus	137-146
26481537-4	2015	METHODS: The mice were sensitized to ovalbumin followed by aerosol allergen challenges and determination of chelidonine"s effect on enhanced pause (Penh), pulmonary eosinophilic infiltration, eotaxin-2, interleukin-4 (IL-4), IL-13, OVA-specific IgE production, and several transcription factors.	chelidonine	108-119	chemokine (C-C motif) ligand 24	Mus musculus	192-201
26481537-5	2015	RESULT: Chelidonine strongly suppressed airway eosinophilia, expression of eotaxin-2, IL-4, and IL-13 cytokine production in bronchoalveolar lavage fluid (BALF).	chelidonine	8-19	chemokine (C-C motif) ligand 24	Mus musculus	75-84
26481537-8	2015	CONCLUSIONS: Chelidonine has profound inhibitory effects on airway inflammation and this effect is caused by suppression of IL-4, eotaxin-2, and OVA-specific IgE production through the STAT6 and Foxp3 pathways.	chelidonine	13-24	chemokine (C-C motif) ligand 24	Mus musculus	130-139
24861132-5	2014	Gene expression profiles of the two Mrc1+ subsets also reveal that Mrc1+GFP(lo) cTMs have a decreased number of immune response genes (Cx3cr1, Lpar6, CD9, Cxcr4, Itga6 and Tgfbetar1), and an increased number of fibrogenic genes (Ltc4s, Retnla, Fgfr1, Mmp9 and Ccl24), consistent with a potential role for cTMs in cardiac fibrosis.	CTMS	80-84	chemokine (C-C motif) ligand 24	Mus musculus	260-265
23574317-11	2013	Administration 2 h prior to ethanol gavage of a neutralizing antibody cocktail to keratinocyte-derived chemokine, macrophage inflammatory protein-2, eotaxin-1 and eotaxin-2 prevented ethanol-induced eosinophil recruitment and airways hyperreactivity.	Ethanol	183-190	chemokine (C-C motif) ligand 24	Mus musculus	163-172
23935198-0	2013	Alendronate attenuates eosinophilic airway inflammation associated with suppression of Th2 cytokines, Th17 cytokines, and eotaxin-2.	Alendronate	0-11	chemokine (C-C motif) ligand 24	Mus musculus	122-131
23238640-6	2013	Chemokines, eotaxin-1 (CCL11) and eotaxin-2 (CCL24), which are known to attract eosinophils, increased in response to halothane treatment.	Halothane	118-127	chemokine (C-C motif) ligand 24	Mus musculus	34-43
23238640-6	2013	Chemokines, eotaxin-1 (CCL11) and eotaxin-2 (CCL24), which are known to attract eosinophils, increased in response to halothane treatment.	Halothane	118-127	chemokine (C-C motif) ligand 24	Mus musculus	45-50
21643893-4	2011	Moreover, dehydroepiandrosterone inhibited chemokines, including CCL11/eotaxin-1 and CCL24/eotaxin-2, and Th2-associated cytokine levels in bronchoalveolar lavage fluid.	Dehydroepiandrosterone	10-32	chemokine (C-C motif) ligand 24	Mus musculus	85-90
22796441-7	2012	Ethanol-gavaged mice had a fivefold increased production of eotaxin-2 (534 pg/mL) and a sevenfold increase in bronchoalveolar eosinophils (70,080 cells).	Ethanol	0-7	chemokine (C-C motif) ligand 24	Mus musculus	60-69
21643893-4	2011	Moreover, dehydroepiandrosterone inhibited chemokines, including CCL11/eotaxin-1 and CCL24/eotaxin-2, and Th2-associated cytokine levels in bronchoalveolar lavage fluid.	Dehydroepiandrosterone	10-32	chemokine (C-C motif) ligand 24	Mus musculus	91-100
19552640-8	2009	In pAMs, IL-4/IL-13 significantly increased eotaxin-2, which was reduced by Mp infection accompanied by dose-dependent PGE(2) induction.	Prostaglandins E	119-122	chemokine (C-C motif) ligand 24	Mus musculus	44-53
19552640-9	2009	Exogenous PGE(2) inhibited IL-4/IL-13-induced eotaxin-2 in a dose-dependent manner.	Prostaglandins E	10-13	chemokine (C-C motif) ligand 24	Mus musculus	46-55
19329284-12	2009	Finally, the skin of AQ-photochallenged site exhibited high expression of CCL24/eotaxin-2, a chemokine for eosinophils.	afloqualone	21-23	chemokine (C-C motif) ligand 24	Mus musculus	74-79
19329284-12	2009	Finally, the skin of AQ-photochallenged site exhibited high expression of CCL24/eotaxin-2, a chemokine for eosinophils.	afloqualone	21-23	chemokine (C-C motif) ligand 24	Mus musculus	80-89
21473902-7	2011	Moreover, PPE-SVC and viscolin inhibited chemokines, including CCL11 and CCL24, and Th2-associated cytokines in bronchoalveolar lavage fluid.	viscolin	22-30	chemokine (C-C motif) ligand 24	Mus musculus	73-78
18981162-6	2008	Using the dextran sodium sulfate (DSS)-induced intestinal epithelial injury model, we show that DSS treatment of mice strongly induced colonic eotaxin-1 and eotaxin-2 expression and eosinophil levels.	dextran sodium sulfate	10-32	chemokine (C-C motif) ligand 24	Mus musculus	157-166
18981162-6	2008	Using the dextran sodium sulfate (DSS)-induced intestinal epithelial injury model, we show that DSS treatment of mice strongly induced colonic eotaxin-1 and eotaxin-2 expression and eosinophil levels.	dss	34-37	chemokine (C-C motif) ligand 24	Mus musculus	157-166
18981162-6	2008	Using the dextran sodium sulfate (DSS)-induced intestinal epithelial injury model, we show that DSS treatment of mice strongly induced colonic eotaxin-1 and eotaxin-2 expression and eosinophil levels.	dss	96-99	chemokine (C-C motif) ligand 24	Mus musculus	157-166
18981162-8	2008	DSS treatment of eotaxin-2(-/-) and eotaxin-1/2(-/-) mice demonstrated that eosinophil recruitment was dependent on eotaxin-1.	dss	0-3	chemokine (C-C motif) ligand 24	Mus musculus	17-26
18981162-8	2008	DSS treatment of eotaxin-2(-/-) and eotaxin-1/2(-/-) mice demonstrated that eosinophil recruitment was dependent on eotaxin-1.	dss	0-3	chemokine (C-C motif) ligand 24	Mus musculus	36-47