PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
28411267-9	2017	Hepatic NFE2 overexpression upregulated miR-423-5p to repress the FAM3A-ATP-Akt pathway, promoting gluconeogenesis and lipid deposition and causing hyperglycemia in normal mice.	Adenosine Triphosphate	72-75	microRNA 423	Mus musculus	40-47
34963484-10	2021	RESULTS: HG treatment increased the cell viability, migration, and angiogenesis accompanied by upregulation of E2F1, miR-423-5p, HIF1alpha, and VEGF levels, but reduction in HIPK2 expression.	Mercury	9-11	microRNA 423	Mus musculus	117-124
34963484-11	2021	Knockdown of E2F1 or miR-423-5p suppressed the HG-induced increases in cell viability, migration, and angiogenesis.	Mercury	47-49	microRNA 423	Mus musculus	21-28
34803150-8	2022	NEAT1 functioned as a molecular sponge of miR-423-5p, and NEAT1 silencing-mediated effects were partly overturned by miR-423-5p interference in HG-induced SV40 MES13 cells.	-5p	49-52	microRNA 423	Mus musculus	117-124