PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
31901745-6	2020	To study this hypothesis, new GABA uptake inhibitors were designed, synthesized, and evaluated in terms of their activity and subtype selectivity for mGAT1-4.	gamma-Aminobutyric Acid	30-34	mannoside acetylglucosaminyltransferase 1	Mus musculus	150-157
34015586-0	2021	Development of tricyclic N-benzyl-4-hydroxybutanamide derivatives as inhibitors of GABA transporters mGAT1-4 with anticonvulsant, antinociceptive, and antidepressant activity.	N-benzyl-4-hydroxybutanamide	25-53	mannoside acetylglucosaminyltransferase 1	Mus musculus	101-108
34015586-0	2021	Development of tricyclic N-benzyl-4-hydroxybutanamide derivatives as inhibitors of GABA transporters mGAT1-4 with anticonvulsant, antinociceptive, and antidepressant activity.	gamma-Aminobutyric Acid	83-87	mannoside acetylglucosaminyltransferase 1	Mus musculus	101-108
34015586-2	2021	Numerous studies have indicated that inhibitors of the GABA transporters mGAT1-4 offer a promising strategy for the treatment of several neurological disorders, including epilepsy, neuropathic pain, and depression.	gamma-Aminobutyric Acid	55-59	mannoside acetylglucosaminyltransferase 1	Mus musculus	73-80
34015586-3	2021	Following our previous results, herein, we report the synthesis, biological evaluation, and structure-activity relationship studies supported by molecular docking and molecular dynamics of a new series of N-benzyl-4-hydroxybutanamide derivatives regarding their inhibitory potency toward mGAT1-4.	N-benzyl-4-hydroxybutanamide	205-233	mannoside acetylglucosaminyltransferase 1	Mus musculus	288-295
33676028-3	2021	Indeed, antisense oligonucleotide (ASO)-mediated knockdown of Mogat1 mRNA, which encodes MGAT1, reduced hepatic MGAT activity and improved glucose tolerance and insulin resistance in high fat diet (HFD) fed mice.	Oligonucleotides	18-33	mannoside acetylglucosaminyltransferase 1	Mus musculus	89-94
33676028-3	2021	Indeed, antisense oligonucleotide (ASO)-mediated knockdown of Mogat1 mRNA, which encodes MGAT1, reduced hepatic MGAT activity and improved glucose tolerance and insulin resistance in high fat diet (HFD) fed mice.	Oligonucleotides, Antisense	35-38	mannoside acetylglucosaminyltransferase 1	Mus musculus	89-94
33716748-7	2021	In addition, subcutaneous BAA-inhibited morphine-induced withdrawal symptoms and CPP expression were totally blocked by the microglial metabolic inhibitor minocycline, dynorphin A antiserum, or specific KOR antagonist GNTI, given intracerebroventricularly.	bulleyaconitine A	26-29	mannoside acetylglucosaminyltransferase 1	Mus musculus	218-222
33716748-7	2021	In addition, subcutaneous BAA-inhibited morphine-induced withdrawal symptoms and CPP expression were totally blocked by the microglial metabolic inhibitor minocycline, dynorphin A antiserum, or specific KOR antagonist GNTI, given intracerebroventricularly.	Morphine	40-48	mannoside acetylglucosaminyltransferase 1	Mus musculus	218-222
32591389-6	2020	Elimination of branched N-glycans in developing B cells via targeted deletion of N-acetylglucosaminyl transferase I (Mgat1) markedly reduced cellularity in the bone marrow and/or spleen and inhibited maturation of pre-, immature, and transitional stage 2 B cells.	n-glycans	24-33	mannoside acetylglucosaminyltransferase 1	Mus musculus	117-122
30957949-1	2019	A new class of nipecotic acid and guvacine derivatives has been synthesized and characterized for their inhibitory potency at mGAT1-4 and binding affinity for mGAT1.	nipecotic acid	15-29	mannoside acetylglucosaminyltransferase 1	Mus musculus	126-133
31071333-2	2019	We showed recently, through genetic ablation of the MGAT1 gene, which encodes an essential glycosyltransferase (GlcNAcT1), that prevention of cardiomyocyte hybrid/complex N-glycosylation was sufficient to cause DCM that led to heart failure and early death.	Nitrogen	115-116	mannoside acetylglucosaminyltransferase 1	Mus musculus	52-57
30957949-1	2019	A new class of nipecotic acid and guvacine derivatives has been synthesized and characterized for their inhibitory potency at mGAT1-4 and binding affinity for mGAT1.	guvacine	34-42	mannoside acetylglucosaminyltransferase 1	Mus musculus	126-133
28197827-9	2017	In addition, phloretin treatment elevated the expression of fatty acid oxidation genes such as carnitine palmitoyltransferase 1a and 1b (Cpt1a and Cpt1b) and reduced expression of monocyte chemoattractant protein-1 (Mcp-1), de novo lipogenesis transcriptional factor peroxisome proliferator-activated receptor-gamma 2 (Ppargamma2), and its target monoacylglycerol O-acyltransferase (Mgat-1) genes.	Phloretin	13-22	mannoside acetylglucosaminyltransferase 1	Mus musculus	383-389
30138037-6	2019	Here, we created a cardiomyocyte-specific Mgat1 knockout (KO) mouse to establish a model useful in exploring the relationship between hybrid/complex N-glycosylation and cardiac function and disease.	Nitrogen	149-150	mannoside acetylglucosaminyltransferase 1	Mus musculus	42-47
29386567-3	2018	MGAT1 is the N-acetylglucosaminyltransferase (GlcNAcT-I) that initiates the synthesis of complex N-glycans.	n-glycans	97-106	mannoside acetylglucosaminyltransferase 1	Mus musculus	0-5
29386567-3	2018	MGAT1 is the N-acetylglucosaminyltransferase (GlcNAcT-I) that initiates the synthesis of complex N-glycans.	n-glycans	97-106	mannoside acetylglucosaminyltransferase 1	Mus musculus	46-55
24973660-1	2014	Six series of 2-substituted 4-aminobutanamide derivatives were synthesized and evaluated for their ability to inhibit GABA transport proteins mGAT1-4 stably expressed in HEK-293 cell lines.	2-substituted 4-aminobutanamide	14-45	mannoside acetylglucosaminyltransferase 1	Mus musculus	142-149
26928671-8	2016	KEY RESULTS: As previously shown for U-50488H, 6"-GNTI markedly increased the threshold for pentylenetetrazole-induced seizures.	3,4-Dichloro-N-methyl-N-(2-(1-pyrrolidinyl)-cyclohexyl)-benzeneacetamide, (trans)-Isomer	37-45	mannoside acetylglucosaminyltransferase 1	Mus musculus	50-54
26928671-8	2016	KEY RESULTS: As previously shown for U-50488H, 6"-GNTI markedly increased the threshold for pentylenetetrazole-induced seizures.	Pentylenetetrazole	92-110	mannoside acetylglucosaminyltransferase 1	Mus musculus	50-54
26834753-6	2015	Mouse MGATs 1 and 2 have been shown to increase oil content transiently in Nicotiana benthamiana leaf tissue by 2.6 fold.	Oils	48-51	mannoside acetylglucosaminyltransferase 1	Mus musculus	6-19
28943619-5	2015	The Acyl-CoA: monoacylglycerolacyltransferase (MGAT) family has three members (MGAT1, -2, and -3) that catalyze the first step in TAG production, conversion of monoacylglycerol (MAG) to diacylglycerol (DAG).	Monoglycerides	14-30	mannoside acetylglucosaminyltransferase 1	Mus musculus	79-96
28943619-5	2015	The Acyl-CoA: monoacylglycerolacyltransferase (MGAT) family has three members (MGAT1, -2, and -3) that catalyze the first step in TAG production, conversion of monoacylglycerol (MAG) to diacylglycerol (DAG).	Monoglycerides	178-181	mannoside acetylglucosaminyltransferase 1	Mus musculus	79-96
28943619-5	2015	The Acyl-CoA: monoacylglycerolacyltransferase (MGAT) family has three members (MGAT1, -2, and -3) that catalyze the first step in TAG production, conversion of monoacylglycerol (MAG) to diacylglycerol (DAG).	Diglycerides	186-200	mannoside acetylglucosaminyltransferase 1	Mus musculus	79-96
28943619-5	2015	The Acyl-CoA: monoacylglycerolacyltransferase (MGAT) family has three members (MGAT1, -2, and -3) that catalyze the first step in TAG production, conversion of monoacylglycerol (MAG) to diacylglycerol (DAG).	Diglycerides	202-205	mannoside acetylglucosaminyltransferase 1	Mus musculus	79-96
25882526-1	2015	A series of beta-amino acids with lipophilic diaromatic side chain was synthesized and characterized pharmacologically on mouse gamma-amino butyric acid (GABA) transporter subtypes mGAT1-4 in order to investigate structure activity relationships (SAR) for mGAT2 (corresponding to hBGT-1).	beta-amino acids	12-28	mannoside acetylglucosaminyltransferase 1	Mus musculus	181-188
24973660-1	2014	Six series of 2-substituted 4-aminobutanamide derivatives were synthesized and evaluated for their ability to inhibit GABA transport proteins mGAT1-4 stably expressed in HEK-293 cell lines.	gamma-Aminobutyric Acid	118-122	mannoside acetylglucosaminyltransferase 1	Mus musculus	142-149
23398473-1	2013	beta-Amino acids sharing a lipophilic diaromatic side chain were synthesized and characterized pharmacologically on mouse GABA transporter subtypes mGAT1-4.	beta-amino acids	0-16	mannoside acetylglucosaminyltransferase 1	Mus musculus	148-155
24503508-1	2014	It has been shown previously that norbinaltorphimine (norBNI) and 5"-guanidinonaltrindole (5"-GNTI), long-acting kappa opioid receptor (KOPR) antagonists, cause frenzied scratching in mice [1,2].	norbinaltorphimine	34-52	mannoside acetylglucosaminyltransferase 1	Mus musculus	94-98
24503508-1	2014	It has been shown previously that norbinaltorphimine (norBNI) and 5"-guanidinonaltrindole (5"-GNTI), long-acting kappa opioid receptor (KOPR) antagonists, cause frenzied scratching in mice [1,2].	17-cyclopropylmethyl-6,7-didehydro-4,5-epoxy-5'-guanidinyl-3,14-dihydroxyindolo(2',3'-6,7)morphinan	66-89	mannoside acetylglucosaminyltransferase 1	Mus musculus	94-98
23775075-0	2013	Functional selectivity of 6"-guanidinonaltrindole (6"-GNTI) at kappa-opioid receptors in striatal neurons.	6'-guanidinonaltrindole	26-49	mannoside acetylglucosaminyltransferase 1	Mus musculus	54-58
23775075-4	2013	Recently, a KOR agonist, 6"-guanidinonaltrindole (6"-GNTI), was shown to display bias toward the activation of G protein-mediated signaling over beta-arrestin2 recruitment.	6'-guanidinonaltrindole	25-48	mannoside acetylglucosaminyltransferase 1	Mus musculus	53-57
22489112-3	2012	All antidepressants tested here inhibited the [(3)H]GABA uptake through mBGT1 and mGATs in a rank order of potency with mBGT1 > mGAT1-3.	gamma-Aminobutyric Acid	52-56	mannoside acetylglucosaminyltransferase 1	Mus musculus	131-138
21629267-2	2011	In mice, N-glycan branching by the Golgi enzymes Mgat1 and/or Mgat5 prevents T cell hyperactivity, cytotoxic T-lymphocyte antigen 4 (CTLA-4) endocytosis, spontaneous inflammatory demyelination and neurodegeneration, the latter pathologies characteristic of MS.	n-glycan	9-17	mannoside acetylglucosaminyltransferase 1	Mus musculus	49-54
23087134-6	2012	The inhibitory potency towards murine plasma membrane GABA transporters (mGAT1-4) was performed as [(3)H]GABA uptake assay based on stably transfected HEK cells.	gamma-Aminobutyric Acid	54-58	mannoside acetylglucosaminyltransferase 1	Mus musculus	73-80
21834068-8	2011	The expression of beta1,2N-acetylglucosaminyltransferase I (GlcNAc-TI) mRNA, which converts high-mannose type N-glycans, was significantly increased in degraded mouse cartilage.	Mannose	97-104	mannoside acetylglucosaminyltransferase 1	Mus musculus	60-69
21834068-8	2011	The expression of beta1,2N-acetylglucosaminyltransferase I (GlcNAc-TI) mRNA, which converts high-mannose type N-glycans, was significantly increased in degraded mouse cartilage.	n-glycans	110-119	mannoside acetylglucosaminyltransferase 1	Mus musculus	60-69
21629267-4	2011	Downregulation of Mgat1 by IL7RA*C and IL2RA*T is opposed by MGAT1 (IV(A)V(T-T)) and vitamin D(3), optimizing branching and mitigating MS risk when combined with enhanced CTLA-4 N-glycosylation by CTLA-4 Thr17.	Vitamin D	85-94	mannoside acetylglucosaminyltransferase 1	Mus musculus	18-23
21629267-4	2011	Downregulation of Mgat1 by IL7RA*C and IL2RA*T is opposed by MGAT1 (IV(A)V(T-T)) and vitamin D(3), optimizing branching and mitigating MS risk when combined with enhanced CTLA-4 N-glycosylation by CTLA-4 Thr17.	Nitrogen	178-179	mannoside acetylglucosaminyltransferase 1	Mus musculus	18-23
17347252-8	2007	Pre-treatment with the kappa selective antagonist GNTI completely blocked the rescue effect of U-50488.	3,4-Dichloro-N-methyl-N-(2-(1-pyrrolidinyl)-cyclohexyl)-benzeneacetamide, (trans)-Isomer	95-102	mannoside acetylglucosaminyltransferase 1	Mus musculus	50-54
20206280-0	2010	Mgat1-dependent N-glycans are essential for the normal development of both vertebrate and invertebrate metazoans.	n-glycans	16-25	mannoside acetylglucosaminyltransferase 1	Mus musculus	0-5
20206280-1	2010	UDP-GlcNAc:alpha3-D-mannoside beta1,2-N-acetylglucosaminyltransferase I (GnTI, encoded by Mgat1) first appeared in evolution at about the same time as metazoa suggesting that GnTI-dependent glycans are essential for the development of multicellular organisms.	Polysaccharides	190-197	mannoside acetylglucosaminyltransferase 1	Mus musculus	0-71
20206280-1	2010	UDP-GlcNAc:alpha3-D-mannoside beta1,2-N-acetylglucosaminyltransferase I (GnTI, encoded by Mgat1) first appeared in evolution at about the same time as metazoa suggesting that GnTI-dependent glycans are essential for the development of multicellular organisms.	Polysaccharides	190-197	mannoside acetylglucosaminyltransferase 1	Mus musculus	73-77
20206280-1	2010	UDP-GlcNAc:alpha3-D-mannoside beta1,2-N-acetylglucosaminyltransferase I (GnTI, encoded by Mgat1) first appeared in evolution at about the same time as metazoa suggesting that GnTI-dependent glycans are essential for the development of multicellular organisms.	Polysaccharides	190-197	mannoside acetylglucosaminyltransferase 1	Mus musculus	90-95
17883406-2	2008	The products of Golgi UDP-GlcNAc:N-acetylglucosaminyltransferases (encoded by Mgat1, Mgat2, Mgat4 and Mgat5) act sequentially to generate the GlcNAc-branched complex-type N-glycans on glycoprotein receptors.	2-acetamido-2-deoxy-4-O-(beta-2-acetamid-2-deoxyglucopyranosyl)glucopyranose	26-32	mannoside acetylglucosaminyltransferase 1	Mus musculus	78-83
17883406-2	2008	The products of Golgi UDP-GlcNAc:N-acetylglucosaminyltransferases (encoded by Mgat1, Mgat2, Mgat4 and Mgat5) act sequentially to generate the GlcNAc-branched complex-type N-glycans on glycoprotein receptors.	n-glycans	171-180	mannoside acetylglucosaminyltransferase 1	Mus musculus	78-83
17883406-3	2008	While elimination of all the branched N-glycans in Mgat1(-/-) mouse embryos is lethal at neural tube fold stage, decreased branching is associated with late developmental defects similar to type 2 of congenital disorders of glycosylation, with developmental and psychomotor abnormalities.	n-glycans	38-47	mannoside acetylglucosaminyltransferase 1	Mus musculus	51-56
17374637-3	2007	However, Mgat1(-/-) eggs with modified N-glycans lacking terminal Gal and GlcNAc residues are fertilized.	n-glycans	39-48	mannoside acetylglucosaminyltransferase 1	Mus musculus	9-14
17374637-3	2007	However, Mgat1(-/-) eggs with modified N-glycans lacking terminal Gal and GlcNAc residues are fertilized.	Galactose	66-69	mannoside acetylglucosaminyltransferase 1	Mus musculus	9-14
17374637-3	2007	However, Mgat1(-/-) eggs with modified N-glycans lacking terminal Gal and GlcNAc residues are fertilized.	Acetylglucosamine	74-80	mannoside acetylglucosaminyltransferase 1	Mus musculus	9-14
9363433-0	1997	Complex N-glycans in Mgat1 null preimplantation embryos arise from maternal Mgat1 RNA.	n-glycans	8-17	mannoside acetylglucosaminyltransferase 1	Mus musculus	21-26
16111674-1	2005	Searching for potent and subtype selective parent structures of the murine gamma-aminobutyric acid (GABA) transporter subtypes mGAT3 and mGAT4 a series of amino acids was characterised in a uniform [3H]GABA uptake test system based on transiently expressed mGAT1-4.	gamma-Aminobutyric Acid	100-104	mannoside acetylglucosaminyltransferase 1	Mus musculus	257-264
9867843-2	1999	Studies on mice with a null mutation in the GnT I gene have indicated that N-glycans play critical roles in mammalian morphogenesis.	n-glycans	75-84	mannoside acetylglucosaminyltransferase 1	Mus musculus	44-49
9363433-0	1997	Complex N-glycans in Mgat1 null preimplantation embryos arise from maternal Mgat1 RNA.	n-glycans	8-17	mannoside acetylglucosaminyltransferase 1	Mus musculus	76-81
9363433-3	1997	However, inner cell mass of all blastocysts from several Mgat1 +/- heterozygous crosses bind the lectin E-PHA, indicating that Mgat1 null mutant blastocysts are able to synthesize complex N-glycans (Campbell et al., (1995) Glycobiology, 5, 535-543).	n-glycans	188-197	mannoside acetylglucosaminyltransferase 1	Mus musculus	127-132
9020882-1	1997	UDP-GlcNAc: alpha-3-D-mannoside beta-1,2-N-acetylglucosaminyltransferase I (EC 2.4.1.101; GlcNAc-T I) is a medial-Golgi enzyme which catalyses the first step in the conversion of oligomannose-type to N-acetyl-lactosamine- and hybrid-type N-glycans and is essential for normal embryogenesis in the mouse.	Uridine Diphosphate N-Acetylglucosamine	0-10	mannoside acetylglucosaminyltransferase 1	Mus musculus	90-100
9020882-1	1997	UDP-GlcNAc: alpha-3-D-mannoside beta-1,2-N-acetylglucosaminyltransferase I (EC 2.4.1.101; GlcNAc-T I) is a medial-Golgi enzyme which catalyses the first step in the conversion of oligomannose-type to N-acetyl-lactosamine- and hybrid-type N-glycans and is essential for normal embryogenesis in the mouse.	alpha-3-d-mannoside	12-31	mannoside acetylglucosaminyltransferase 1	Mus musculus	90-100
9020882-1	1997	UDP-GlcNAc: alpha-3-D-mannoside beta-1,2-N-acetylglucosaminyltransferase I (EC 2.4.1.101; GlcNAc-T I) is a medial-Golgi enzyme which catalyses the first step in the conversion of oligomannose-type to N-acetyl-lactosamine- and hybrid-type N-glycans and is essential for normal embryogenesis in the mouse.	oligomannose	179-191	mannoside acetylglucosaminyltransferase 1	Mus musculus	90-100
9020882-1	1997	UDP-GlcNAc: alpha-3-D-mannoside beta-1,2-N-acetylglucosaminyltransferase I (EC 2.4.1.101; GlcNAc-T I) is a medial-Golgi enzyme which catalyses the first step in the conversion of oligomannose-type to N-acetyl-lactosamine- and hybrid-type N-glycans and is essential for normal embryogenesis in the mouse.	N-acetyllactosamine	200-220	mannoside acetylglucosaminyltransferase 1	Mus musculus	90-100
9020882-1	1997	UDP-GlcNAc: alpha-3-D-mannoside beta-1,2-N-acetylglucosaminyltransferase I (EC 2.4.1.101; GlcNAc-T I) is a medial-Golgi enzyme which catalyses the first step in the conversion of oligomannose-type to N-acetyl-lactosamine- and hybrid-type N-glycans and is essential for normal embryogenesis in the mouse.	n-glycans	238-247	mannoside acetylglucosaminyltransferase 1	Mus musculus	90-100
7881185-2	1994	A key transferase in the generation of mature N-linked carbohydrates is the medial Golgi enzyme N-acetylglucosaminyltransferase I (EC 2.4.1.101; GlcNAc-TI) which is encoded by the Mgat-1 gene.	n-linked carbohydrates	46-68	mannoside acetylglucosaminyltransferase 1	Mus musculus	145-154
8956109-8	1996	Although mGATs 1 and 4 may subserve the classical purpose of terminating inhibitory GABAergic transmission through neuronal and glial uptake mechanisms, GABA transporters in the pia-arachnoid may help to regulate the amount of GABA available to proliferating and migrating neurons at the sub-pial surface during perinatal development.	gamma-Aminobutyric Acid	84-88	mannoside acetylglucosaminyltransferase 1	Mus musculus	9-22
8855305-1	1996	Mice lacking the complex subset of N-glycans due to inactivation of the Mgat1 gene die at mid-gestation, making it difficult to identify specific biological functions for this class of cell surface carbohydrates.	n-glycans	35-44	mannoside acetylglucosaminyltransferase 1	Mus musculus	72-77
8855305-3	1996	The Mgat1 gene encodes N-acetylglucosaminyltransferase I (Glc-NAc-TI; EC 2.4.1.101), the transferase that initiates the synthesis of complex N-glycans.	n-glycans	141-150	mannoside acetylglucosaminyltransferase 1	Mus musculus	4-9
8855305-3	1996	The Mgat1 gene encodes N-acetylglucosaminyltransferase I (Glc-NAc-TI; EC 2.4.1.101), the transferase that initiates the synthesis of complex N-glycans.	n-glycans	141-150	mannoside acetylglucosaminyltransferase 1	Mus musculus	58-68
8563140-0	1995	Complex asparagine-linked oligosaccharides in Mgat1-null embryos.	asparagine-linked oligosaccharides	8-42	mannoside acetylglucosaminyltransferase 1	Mus musculus	46-51
8563140-1	1995	To investigate the developmental role of complex N-linked oligosaccharides, we previously inactivated the mouse Mgat1 gene which encodes UDP-N-acetylglucosamine: alpha-3-D-mannoside beta-1,2-N-acetylglucosaminyltransferase I (GlcNAc-TI).	n-linked oligosaccharides	49-74	mannoside acetylglucosaminyltransferase 1	Mus musculus	112-117
8563140-1	1995	To investigate the developmental role of complex N-linked oligosaccharides, we previously inactivated the mouse Mgat1 gene which encodes UDP-N-acetylglucosamine: alpha-3-D-mannoside beta-1,2-N-acetylglucosaminyltransferase I (GlcNAc-TI).	Uridine Diphosphate N-Acetylglucosamine	137-160	mannoside acetylglucosaminyltransferase 1	Mus musculus	112-117
8563140-6	1995	However, apparently normal levels of complex N-glycans were observed in E3.5 pre-implantation Mgat1-null embryos prior to declining to undetectable levels by E7.5.	n-glycans	45-54	mannoside acetylglucosaminyltransferase 1	Mus musculus	94-99
8563140-8	1995	Mgat1-null embryos appear to complete pre-implantation development in the presence of maternally derived complex N-glycans, and may die at later stages, post E7.5, when a requirement for embryonically derived complex N-glycans arises.	n-glycans	113-122	mannoside acetylglucosaminyltransferase 1	Mus musculus	0-5
8563140-8	1995	Mgat1-null embryos appear to complete pre-implantation development in the presence of maternally derived complex N-glycans, and may die at later stages, post E7.5, when a requirement for embryonically derived complex N-glycans arises.	n-glycans	217-226	mannoside acetylglucosaminyltransferase 1	Mus musculus	0-5
7881185-2	1994	A key transferase in the generation of mature N-linked carbohydrates is the medial Golgi enzyme N-acetylglucosaminyltransferase I (EC 2.4.1.101; GlcNAc-TI) which is encoded by the Mgat-1 gene.	n-linked carbohydrates	46-68	mannoside acetylglucosaminyltransferase 1	Mus musculus	180-186
7881185-12	1994	The combined data suggest that 5" exons in the Mgat-1 gene are differentially utilized by tissue-specific promoters and that transcription factor(s) which specify production of the approximately 3.3 kb Mgat-1 mRNA are induced by retinoic acid treatment of P19 EC cells.	Tretinoin	229-242	mannoside acetylglucosaminyltransferase 1	Mus musculus	47-53
7881185-12	1994	The combined data suggest that 5" exons in the Mgat-1 gene are differentially utilized by tissue-specific promoters and that transcription factor(s) which specify production of the approximately 3.3 kb Mgat-1 mRNA are induced by retinoic acid treatment of P19 EC cells.	Tretinoin	229-242	mannoside acetylglucosaminyltransferase 1	Mus musculus	202-208
8187759-3	1994	Complex N-linked oligosaccharide biosynthesis is initiated in the Golgi apparatus by the action of Mgat-1-encoded UDP-N-acetylglucosamine:alpha-3-D- mannoside beta-1,2-N-acetylglucosaminyltransferase I (GlcNAc-TI).	n-linked oligosaccharide	8-32	mannoside acetylglucosaminyltransferase 1	Mus musculus	99-105
8187759-3	1994	Complex N-linked oligosaccharide biosynthesis is initiated in the Golgi apparatus by the action of Mgat-1-encoded UDP-N-acetylglucosamine:alpha-3-D- mannoside beta-1,2-N-acetylglucosaminyltransferase I (GlcNAc-TI).	Uridine Diphosphate N-Acetylglucosamine	114-137	mannoside acetylglucosaminyltransferase 1	Mus musculus	99-105
8187759-3	1994	Complex N-linked oligosaccharide biosynthesis is initiated in the Golgi apparatus by the action of Mgat-1-encoded UDP-N-acetylglucosamine:alpha-3-D- mannoside beta-1,2-N-acetylglucosaminyltransferase I (GlcNAc-TI).	n-linked oligosaccharide	8-32	mannoside acetylglucosaminyltransferase 1	Mus musculus	203-212
8187759-3	1994	Complex N-linked oligosaccharide biosynthesis is initiated in the Golgi apparatus by the action of Mgat-1-encoded UDP-N-acetylglucosamine:alpha-3-D- mannoside beta-1,2-N-acetylglucosaminyltransferase I (GlcNAc-TI).	Uridine Diphosphate N-Acetylglucosamine	114-137	mannoside acetylglucosaminyltransferase 1	Mus musculus	203-212
8187759-5	1994	Inactivation of both Mgat-1 alleles produced deficiencies in GlcNAc-TI activity and complex N-linked oligosaccharides.	n-linked oligosaccharides	92-117	mannoside acetylglucosaminyltransferase 1	Mus musculus	21-27
8163508-11	1994	Furthermore, GnTI hybrid molecules showing increased cell surface expression were more readily extracted in a low salt buffer, suggesting that Golgi localized GnTI differs in physicochemical properties from cell surface GnTI.	Salts	114-118	mannoside acetylglucosaminyltransferase 1	Mus musculus	13-17
8187759-8	1994	Complex N-linked oligosaccharides are important for morphogenic processes as neural tube formation, vascularization and the determination of left-right body plan asymmetry were impaired in the absence of a functional Mgat-1 gene.	n-linked oligosaccharides	8-33	mannoside acetylglucosaminyltransferase 1	Mus musculus	217-223
8163508-11	1994	Furthermore, GnTI hybrid molecules showing increased cell surface expression were more readily extracted in a low salt buffer, suggesting that Golgi localized GnTI differs in physicochemical properties from cell surface GnTI.	Salts	114-118	mannoside acetylglucosaminyltransferase 1	Mus musculus	159-163
8163508-11	1994	Furthermore, GnTI hybrid molecules showing increased cell surface expression were more readily extracted in a low salt buffer, suggesting that Golgi localized GnTI differs in physicochemical properties from cell surface GnTI.	Salts	114-118	mannoside acetylglucosaminyltransferase 1	Mus musculus	159-163
34505434-21	2021	Based on the upregulation of UDP-GlcNAc under hepatocyte LAMTOR1 depletion, it was predicted that LAMTOR1 positively influenced MGAT1 (R=0.47), a gene encoding alpha-1,3-mannosyl-glycoprotein 2-beta-N-acetylglucosaminyltransferase.	Uridine Diphosphate N-Acetylglucosamine	29-39	mannoside acetylglucosaminyltransferase 1	Mus musculus	128-133
1447191-1	1992	UDP-GlcNAc:alpha 3-D-mannoside beta 1,2-N-acetylglucosaminyltransferase I (GnTI) is an N(in)/C(out) (type II) membrane protein, localized in the medial-Golgi, that initiates the conversion of high mannose N-glycans to complex N-glycans.	Mannose	197-204	mannoside acetylglucosaminyltransferase 1	Mus musculus	0-73
1447191-1	1992	UDP-GlcNAc:alpha 3-D-mannoside beta 1,2-N-acetylglucosaminyltransferase I (GnTI) is an N(in)/C(out) (type II) membrane protein, localized in the medial-Golgi, that initiates the conversion of high mannose N-glycans to complex N-glycans.	Mannose	197-204	mannoside acetylglucosaminyltransferase 1	Mus musculus	75-79
1447191-1	1992	UDP-GlcNAc:alpha 3-D-mannoside beta 1,2-N-acetylglucosaminyltransferase I (GnTI) is an N(in)/C(out) (type II) membrane protein, localized in the medial-Golgi, that initiates the conversion of high mannose N-glycans to complex N-glycans.	n-glycans	205-214	mannoside acetylglucosaminyltransferase 1	Mus musculus	0-73
1447191-1	1992	UDP-GlcNAc:alpha 3-D-mannoside beta 1,2-N-acetylglucosaminyltransferase I (GnTI) is an N(in)/C(out) (type II) membrane protein, localized in the medial-Golgi, that initiates the conversion of high mannose N-glycans to complex N-glycans.	n-glycans	205-214	mannoside acetylglucosaminyltransferase 1	Mus musculus	75-79
1447191-1	1992	UDP-GlcNAc:alpha 3-D-mannoside beta 1,2-N-acetylglucosaminyltransferase I (GnTI) is an N(in)/C(out) (type II) membrane protein, localized in the medial-Golgi, that initiates the conversion of high mannose N-glycans to complex N-glycans.	n-glycans	226-235	mannoside acetylglucosaminyltransferase 1	Mus musculus	0-73
1447191-1	1992	UDP-GlcNAc:alpha 3-D-mannoside beta 1,2-N-acetylglucosaminyltransferase I (GnTI) is an N(in)/C(out) (type II) membrane protein, localized in the medial-Golgi, that initiates the conversion of high mannose N-glycans to complex N-glycans.	n-glycans	226-235	mannoside acetylglucosaminyltransferase 1	Mus musculus	75-79
1447191-8	1992	The ovalbumin/GnTI hybrid molecules in the transfected L cells were N-glycosylated, indicating an N(in)/C(out) membrane orientation, and were localized by immunoperoxidase electron microscopy to one or two cisternae of the medial-Golgi (90% of stained Golgi profiles showed medial-cisternae staining).	Nitrogen	68-69	mannoside acetylglucosaminyltransferase 1	Mus musculus	14-18
1533386-4	1992	UDP-N-acetylglucosamine:alpha-3-D-mannoside beta-1,2-N-acetylglucosaminyltransferase I (EC 2.4.1.101; GlcNAc-TI) is a key enzyme in the process because it is essential for the conversion of high-mannose N-glycans to complex and hybrid N-glycans.	Uridine Diphosphate N-Acetylglucosamine	0-23	mannoside acetylglucosaminyltransferase 1	Mus musculus	102-111
1533386-4	1992	UDP-N-acetylglucosamine:alpha-3-D-mannoside beta-1,2-N-acetylglucosaminyltransferase I (EC 2.4.1.101; GlcNAc-TI) is a key enzyme in the process because it is essential for the conversion of high-mannose N-glycans to complex and hybrid N-glycans.	alpha-3-d-mannoside beta-	24-49	mannoside acetylglucosaminyltransferase 1	Mus musculus	102-111
1533386-4	1992	UDP-N-acetylglucosamine:alpha-3-D-mannoside beta-1,2-N-acetylglucosaminyltransferase I (EC 2.4.1.101; GlcNAc-TI) is a key enzyme in the process because it is essential for the conversion of high-mannose N-glycans to complex and hybrid N-glycans.	mannose n-glycans	195-212	mannoside acetylglucosaminyltransferase 1	Mus musculus	102-111
1533386-4	1992	UDP-N-acetylglucosamine:alpha-3-D-mannoside beta-1,2-N-acetylglucosaminyltransferase I (EC 2.4.1.101; GlcNAc-TI) is a key enzyme in the process because it is essential for the conversion of high-mannose N-glycans to complex and hybrid N-glycans.	n-glycans	203-212	mannoside acetylglucosaminyltransferase 1	Mus musculus	102-111
34505434-21	2021	Based on the upregulation of UDP-GlcNAc under hepatocyte LAMTOR1 depletion, it was predicted that LAMTOR1 positively influenced MGAT1 (R=0.47), a gene encoding alpha-1,3-mannosyl-glycoprotein 2-beta-N-acetylglucosaminyltransferase.	Uridine Diphosphate N-Acetylglucosamine	29-39	mannoside acetylglucosaminyltransferase 1	Mus musculus	160-230