PMID-sentid Pub_year Sent_text comp_official_name comp_offset protein_name organism prot_offset 35302183-0 2022 Effects of SIDT2 on the miR-25/NOX4/HuR axis and SIRT3 mRNA stability lead to ROS-mediated TNF-alpha expression in hydroquinone-treated leukemia cells. ros 78-81 microRNA 25 Homo sapiens 24-30 35194770-3 2022 METHODS: IL-1beta and two additional ER stress activators, palmitate and tunicamycin were applied to evaluate the expression level miR-25 by Taqman RT-PCR. Palmitates 59-68 microRNA 25 Homo sapiens 131-137 35194770-3 2022 METHODS: IL-1beta and two additional ER stress activators, palmitate and tunicamycin were applied to evaluate the expression level miR-25 by Taqman RT-PCR. Tunicamycin 73-84 microRNA 25 Homo sapiens 131-137 35613957-13 2022 The potential to detect PC was good for a combination of PJ EV-miR-21, EV-miR-25, EV-miR-16 and serum miR-210, CA-19-9, with an area under the curve of 0.91, a specificity of 84.2% and a sensitivity of 81.5%. .alpha.-Glutamylvaline 71-73 microRNA 25 Homo sapiens 74-80 35302183-10 2022 Overall, these results indicate that SIDT2 regulates the miR-25/NOX4/HuR axis and SIRT3 mRNA destabilization, leading to ROS-mediated TNF-alpha upregulation in HQ-treated U937 cells. ros 121-124 microRNA 25 Homo sapiens 57-63 35302183-0 2022 Effects of SIDT2 on the miR-25/NOX4/HuR axis and SIRT3 mRNA stability lead to ROS-mediated TNF-alpha expression in hydroquinone-treated leukemia cells. hydroquinone 115-127 microRNA 25 Homo sapiens 24-30 33253979-8 2021 Furthermore, miR-21, miR-25, miR-27b, miR-19b, miR-125b, miR-146a and miR-210 predicted response to platinum based treatments. Platinum 100-108 microRNA 25 Homo sapiens 21-27 34983615-12 2022 Using RNA and microRNA sequencing, we identified miR-551b and miR-25 as important miRs mediating Dex resistance in MM. Dexamethasone 97-100 microRNA 25 Homo sapiens 62-68 34983615-13 2022 Overexpression of miR-551b and miR-25 caused resistance to Dex, however, knockdown of miR-551b and miR-25 significantly enhanced Dex sensitivity in MM. Dexamethasone 59-62 microRNA 25 Homo sapiens 31-37 34983615-13 2022 Overexpression of miR-551b and miR-25 caused resistance to Dex, however, knockdown of miR-551b and miR-25 significantly enhanced Dex sensitivity in MM. Dexamethasone 129-132 microRNA 25 Homo sapiens 99-105 34983615-14 2022 SAE2 knockdown or TAK-981 treatment downregulated the expression of miR-551b and miR-25, leading to induction of miR targets ZFP36, ULK1 and p27, resulting in apoptosis and autophagy. TAK-981 18-25 microRNA 25 Homo sapiens 81-87 34012877-0 2021 Role of microRNA-25 in high glucose cultured Muller glia. Glucose 28-35 microRNA 25 Homo sapiens 8-19 34012877-1 2021 AIM: To investigate the role of microRNA-25 (miR-25) in proliferation and apoptosis of retinal Muller glia (MG) under high glucose condition. Glucose 123-130 microRNA 25 Homo sapiens 32-43 34012877-1 2021 AIM: To investigate the role of microRNA-25 (miR-25) in proliferation and apoptosis of retinal Muller glia (MG) under high glucose condition. Glucose 123-130 microRNA 25 Homo sapiens 45-51 34012877-3 2021 The expression level of miR-25 under normal and high glucose conditions were validated by quantitative reverse transcription polymerase chain reaction (RT-qPCR). Glucose 53-60 microRNA 25 Homo sapiens 24-30 34012877-5 2021 RESULTS: Immunocytochemistry and flow cytometry confirmed the high purity of primary cultured MG. RT-PCR results showed that the expression level of miR-25 was significantly repressed in HGMG, while over-expression of miR-25 by miR-25 mimic markedly inhibited the high glucose induced cell apoptosis and promoted the proliferation of MG. hgmg 187-191 microRNA 25 Homo sapiens 149-155 34012877-5 2021 RESULTS: Immunocytochemistry and flow cytometry confirmed the high purity of primary cultured MG. RT-PCR results showed that the expression level of miR-25 was significantly repressed in HGMG, while over-expression of miR-25 by miR-25 mimic markedly inhibited the high glucose induced cell apoptosis and promoted the proliferation of MG. Glucose 269-276 microRNA 25 Homo sapiens 149-155 34012877-5 2021 RESULTS: Immunocytochemistry and flow cytometry confirmed the high purity of primary cultured MG. RT-PCR results showed that the expression level of miR-25 was significantly repressed in HGMG, while over-expression of miR-25 by miR-25 mimic markedly inhibited the high glucose induced cell apoptosis and promoted the proliferation of MG. Magnesium 94-96 microRNA 25 Homo sapiens 149-155 34012877-6 2021 CONCLUSION: The expression level of miR-25 is significantly downregulated in HGMG and its overexpression could attenuate the high glucose damages on MG by promoting proliferation and reducing apoptosis. Glucose 130-137 microRNA 25 Homo sapiens 36-42 34012877-6 2021 CONCLUSION: The expression level of miR-25 is significantly downregulated in HGMG and its overexpression could attenuate the high glucose damages on MG by promoting proliferation and reducing apoptosis. Magnesium 79-81 microRNA 25 Homo sapiens 36-42 35165511-0 2022 MiR-25 enhances autophagy and promotes sorafenib resistance of hepatocellular carcinoma via targeting FBXW7. Sorafenib 39-48 microRNA 25 Homo sapiens 0-6 35165511-9 2022 MiR-25 enhanced sorafenib resistance of HCC cells and autophagy. Sorafenib 16-25 microRNA 25 Homo sapiens 0-6 35165511-11 2022 Overexpression of FBXW7 could reverse the increase of sorafenib resistance caused by miR-25 mimics. Sorafenib 54-63 microRNA 25 Homo sapiens 85-91 35165511-12 2022 Our results suggested that miR-25 increased the sorafenib resistance of HCC via inducing autophagy. Sorafenib 48-57 microRNA 25 Homo sapiens 27-33 33549596-8 2021 miR-25 in the cancer cell-secreted MVs was transferred to their host cells HepG2 and Huh7 cells and reversed the sorafenib induced expression of tumor suppressor p53. Sorafenib 113-122 microRNA 25 Homo sapiens 0-6 32663850-7 2021 PegIFN-alpha treatment upregulated exosomal hsa-miR-193a-5p, hsa-miR-25-5p, and hsa-miR-574-5p that could partially inhibit HBV replication and transcription. pegifn-alpha 0-12 microRNA 25 Homo sapiens 61-71 27840896-5 2016 Furthermore, we found that knockdown of miR-25 by its antisense oligonucleotide (anti-miR-25) significantly increased the sensitivity of LCSCs to TRAIL-induced apoptosis. Oligonucleotides 64-79 microRNA 25 Homo sapiens 40-46 31431131-0 2019 Epigallocatechin gallate (EGCG) suppresses growth and tumorigenicity in breast cancer cells by downregulation of miR-25. epigallocatechin gallate 0-24 microRNA 25 Homo sapiens 113-119 31431131-0 2019 Epigallocatechin gallate (EGCG) suppresses growth and tumorigenicity in breast cancer cells by downregulation of miR-25. epigallocatechin gallate 26-30 microRNA 25 Homo sapiens 113-119 31431131-4 2019 Moreover, EGCG inhibited miR-25 expression and increased PARP, pro-caspase-3 and pro-caspase-9 at protein levels. epigallocatechin gallate 10-14 microRNA 25 Homo sapiens 25-31 31431131-5 2019 Restoration of miR-25 inhibited EGCG-induced cell apoptosis. epigallocatechin gallate 32-36 microRNA 25 Homo sapiens 15-21 31431131-6 2019 Furthermore, EGCG suppressed tumor growth in vivo by downregulating the expression of miR-25 and proteins associated with apoptosis, which was further confirmed by a reduction of Ki-67 and increase of pro-apoptotic PARP expression as determined by immunohistochemistry staining. epigallocatechin gallate 13-17 microRNA 25 Homo sapiens 86-92 31015415-0 2019 Excessive miR-25-3p maturation via N6-methyladenosine stimulated by cigarette smoke promotes pancreatic cancer progression. p-Bis(2-chloroethyl)amino-o-methoxyphenylalanine 17-19 microRNA 25 Homo sapiens 10-16 31015415-0 2019 Excessive miR-25-3p maturation via N6-methyladenosine stimulated by cigarette smoke promotes pancreatic cancer progression. N-methyladenosine 35-53 microRNA 25 Homo sapiens 10-16 28968550-8 2017 Expression levels of miR-25 and miR-210 were significantly higher, and those of PTEN and AIFM3 significantly lower, in c-EV-treated versus medium-only or EV-depleted control groups. c-ev 119-123 microRNA 25 Homo sapiens 21-27 27911275-0 2017 microRNA-25 targets PKCzeta and protects osteoblastic cells from dexamethasone via activating AMPK signaling. Dexamethasone 65-78 microRNA 25 Homo sapiens 0-11 27911275-4 2017 Forced-expression of miR-25 downregulated PKCzeta and activated AMPK in human osteoblastic cells (OB-6 and hFOB1.19 lines), which thereafter protected cells from Dex. Dexamethasone 162-165 microRNA 25 Homo sapiens 21-27 27911275-9 2017 Further studies showed that miR-25 expression increased NADPH activity and suppressed Dex-induced oxidative stress in osteoblastic cells. NADP 56-61 microRNA 25 Homo sapiens 28-34 27911275-9 2017 Further studies showed that miR-25 expression increased NADPH activity and suppressed Dex-induced oxidative stress in osteoblastic cells. Dexamethasone 86-89 microRNA 25 Homo sapiens 28-34 27911275-12 2017 These results suggest that miR-25-5p targets PKCzeta and protects osteoblastic cells from Dex possibly via activating AMPK signaling. Dexamethasone 90-93 microRNA 25 Homo sapiens 27-33 33344223-12 2020 Thus, melatonin exerted anti-proliferative, anti-invasive, and anti-migrative effects on OSCC via miR-25-5p/NEDD9 pathway. Melatonin 6-15 microRNA 25 Homo sapiens 98-104 31298363-17 2019 Microarray and qRT-PCR showed that miR-25 expression was downregulated by PG treatment. physcion 8-O-glucopyranoside 74-76 microRNA 25 Homo sapiens 35-41 31298363-18 2019 Moreover, our results indicated that the anti-cancer activities of PG were augmented by miR-25 knockdown and attenuated by ectopic miR-25 expression. physcion 8-O-glucopyranoside 67-69 microRNA 25 Homo sapiens 88-94 31298363-18 2019 Moreover, our results indicated that the anti-cancer activities of PG were augmented by miR-25 knockdown and attenuated by ectopic miR-25 expression. physcion 8-O-glucopyranoside 67-69 microRNA 25 Homo sapiens 131-137 30190521-6 2019 Among miRNA, combination of miR-1290, miR-196b and miR-135a in tumor tissue, and miR-21, miR-25, miR27b, and miR-326 in plasma were predictive for response to platinum-based chemotherapy in advanced NSCLC. Platinum 159-167 microRNA 25 Homo sapiens 89-95 30147021-9 2019 The findings further suggest that the upregulation of miR-25 and miR-181 family members in SCA3- LCs reflects a cell type-specific, protective mechanism to diminish polyQ-mediated cytotoxic effects. polyglutamine 165-170 microRNA 25 Homo sapiens 54-60 30302568-0 2018 A voltammetric assay for microRNA-25 based on the use of amino-functionalized graphene quantum dots and ss- and ds-DNAs as gene probes. Graphite 78-86 microRNA 25 Homo sapiens 25-36 30302568-10 2018 Graphical abstract A novel electrochemical nanogenosensor is introduced for simple and sensitive determination of microRNA-25, as a biomarker, based on amino-functionalized graphene quantum dots (as a surface modifier) and p-biphenol (as an electroactive label). Graphite 173-181 microRNA 25 Homo sapiens 114-125 29765562-6 2018 Many of miR-25 target mRNAs are involved in biological processes such as cell proliferation, differentiation, and migration, apoptosis, oxidative stress, inflammation, calcium handling, etc. Calcium 168-175 microRNA 25 Homo sapiens 8-14 29113252-3 2017 The present study aimed to investigate the potential role of miR-25 in the cisplatin sensitivity of GC cells. Cisplatin 75-84 microRNA 25 Homo sapiens 61-67 29113252-4 2017 The expression level of miR-25 was significantly upregulated in the cisplatin-resistant GC cell line SGC-7901/DDP compared with the SGC-7901 parental cell line. Cisplatin 68-77 microRNA 25 Homo sapiens 24-30 29113252-5 2017 Overexpression of miR-25 significantly enhanced cell cycle progression and decreased the sensitivity of SGC-7901 cells to cisplatin, whereas inhibition of miR-25 in the SGC-7901/DDP cisplatin-resistant cells resulted in cell cycle arrest at the G0/G1 phase and significantly increased drug sensitivity. Cisplatin 122-131 microRNA 25 Homo sapiens 18-24 29113252-7 2017 These findings suggest that upregulation of miR-25 is important for GC cells to establish a cisplatin-resistant phenotype via a FOXO3a-dependent mechanism. Cisplatin 92-101 microRNA 25 Homo sapiens 44-50 29113252-8 2017 Therefore, targeting miR-25 may be a promising therapeutic approach to treat patients with cisplatin-resistant GC. Cisplatin 91-100 microRNA 25 Homo sapiens 21-27 27840896-5 2016 Furthermore, we found that knockdown of miR-25 by its antisense oligonucleotide (anti-miR-25) significantly increased the sensitivity of LCSCs to TRAIL-induced apoptosis. Oligonucleotides 64-79 microRNA 25 Homo sapiens 86-92 27103406-2 2016 In this review we have focused on three groups of miRNAs: (a) miRNAs that are modulated both by ROS and Cai: miR-181a and miR-205; (b) miRNAs that are modulated by ROS and have an effect on Cai: miR-1, miR-21, miR-24, miR-25, miR-185 and miR-214; (c) miRNAs that modulate both ROS and Cai: miR-133; miR-145, miR-495, and we have analyzed their effects on cell signaling and cell function. Reactive Oxygen Species 164-167 microRNA 25 Homo sapiens 218-224 28101226-13 2016 These results indicate that overexpression of plasma miR-25, miR-21, miR-27b and miR-326, prior to treatment, in patients with advanced LAC is predictive of non-benefit from first-line pemetrexed and platinum-based chemotherapy, and is associated with decreased PFS. Pemetrexed 185-195 microRNA 25 Homo sapiens 53-59 28101226-13 2016 These results indicate that overexpression of plasma miR-25, miR-21, miR-27b and miR-326, prior to treatment, in patients with advanced LAC is predictive of non-benefit from first-line pemetrexed and platinum-based chemotherapy, and is associated with decreased PFS. Platinum 200-208 microRNA 25 Homo sapiens 53-59 27103406-2 2016 In this review we have focused on three groups of miRNAs: (a) miRNAs that are modulated both by ROS and Cai: miR-181a and miR-205; (b) miRNAs that are modulated by ROS and have an effect on Cai: miR-1, miR-21, miR-24, miR-25, miR-185 and miR-214; (c) miRNAs that modulate both ROS and Cai: miR-133; miR-145, miR-495, and we have analyzed their effects on cell signaling and cell function. Reactive Oxygen Species 96-99 microRNA 25 Homo sapiens 218-224 27103406-2 2016 In this review we have focused on three groups of miRNAs: (a) miRNAs that are modulated both by ROS and Cai: miR-181a and miR-205; (b) miRNAs that are modulated by ROS and have an effect on Cai: miR-1, miR-21, miR-24, miR-25, miR-185 and miR-214; (c) miRNAs that modulate both ROS and Cai: miR-133; miR-145, miR-495, and we have analyzed their effects on cell signaling and cell function. Reactive Oxygen Species 164-167 microRNA 25 Homo sapiens 218-224 26687391-8 2016 The PFS times among patients in the pemetrexed group varied significantly and were related to patient expression levels of miR-25, miR-145, and miR-210, whereas patients in the observation group showed no differences in PFS time. Pemetrexed 36-46 microRNA 25 Homo sapiens 123-129 27311985-8 2016 Transfection of HepaRG cells with hsa-miR-25-3p mimics inhibited expression of the endogenous CYP2B6 gene and it also decreased rifampicin-dependent induction of CYP2B6 at the mRNA and protein levels. Rifampin 128-138 microRNA 25 Homo sapiens 34-44 26687391-9 2016 Our data suggest miR-25, miR-145, and miR-210 as predictors for the efficacy of maintenance treatment with pemetrexed in lung adenocarcinoma patients who were negative for EGFR mutations or ALK translocations. Pemetrexed 107-117 microRNA 25 Homo sapiens 17-23 26315342-5 2016 Moreover, restoration of miR-25 promoted the H2O2-induced melanocyte destruction and led to the dysfunction of melanocytes. Hydrogen Peroxide 45-49 microRNA 25 Homo sapiens 25-31 25432132-8 2015 Downregulation of miR-25 markedly inhibited A549 cell proliferation, induced G1 cell cycle arrest, increased cisplatin sensitivity, and suppressed the growth of caner cell xenograft in vivo. Cisplatin 109-118 microRNA 25 Homo sapiens 18-24 24798859-5 2015 Here, we report that the environmental carcinogen benzo[a]pyrene (BaP) upregulated the expression of seven p53-targeting miRNAs (miR-25, miR-15a, miR-16, miR-92, miR-125b, miR-141, and miR-200a), while 2,3,7,8-tetrachlorodibenzo-rho-dioxin (TCDD) upregulated two of them (miR-25 and miR-92) in MM cells. Benzo(a)pyrene 50-64 microRNA 25 Homo sapiens 272-278 24798859-5 2015 Here, we report that the environmental carcinogen benzo[a]pyrene (BaP) upregulated the expression of seven p53-targeting miRNAs (miR-25, miR-15a, miR-16, miR-92, miR-125b, miR-141, and miR-200a), while 2,3,7,8-tetrachlorodibenzo-rho-dioxin (TCDD) upregulated two of them (miR-25 and miR-92) in MM cells. Benzo(a)pyrene 66-69 microRNA 25 Homo sapiens 129-135 24798859-5 2015 Here, we report that the environmental carcinogen benzo[a]pyrene (BaP) upregulated the expression of seven p53-targeting miRNAs (miR-25, miR-15a, miR-16, miR-92, miR-125b, miR-141, and miR-200a), while 2,3,7,8-tetrachlorodibenzo-rho-dioxin (TCDD) upregulated two of them (miR-25 and miR-92) in MM cells. Benzo(a)pyrene 66-69 microRNA 25 Homo sapiens 272-278 24798859-6 2015 The miR-25 promoter was activated by both BaP and TCDD, and this response was mediated by the aryl hydrocarbon receptor (AhR). Benzo(a)pyrene 42-45 microRNA 25 Homo sapiens 4-10 24798859-6 2015 The miR-25 promoter was activated by both BaP and TCDD, and this response was mediated by the aryl hydrocarbon receptor (AhR). Polychlorinated Dibenzodioxins 50-54 microRNA 25 Homo sapiens 4-10 24798859-8 2015 We found that (-)-epigallocatechin-3-gallate (EGCG), a constituent of green tea and a major component of the botanical drug Polyphenon E, reduced the expression of four p53-targeting miRNAs, including miR-25, miR-92, miR-141, and miR-200a. epigallocatechin gallate 14-44 microRNA 25 Homo sapiens 202-208 24798859-8 2015 We found that (-)-epigallocatechin-3-gallate (EGCG), a constituent of green tea and a major component of the botanical drug Polyphenon E, reduced the expression of four p53-targeting miRNAs, including miR-25, miR-92, miR-141, and miR-200a. epigallocatechin gallate 46-50 microRNA 25 Homo sapiens 202-208 24798859-8 2015 We found that (-)-epigallocatechin-3-gallate (EGCG), a constituent of green tea and a major component of the botanical drug Polyphenon E, reduced the expression of four p53-targeting miRNAs, including miR-25, miR-92, miR-141, and miR-200a. polyphenon E 124-137 microRNA 25 Homo sapiens 202-208 24798859-5 2015 Here, we report that the environmental carcinogen benzo[a]pyrene (BaP) upregulated the expression of seven p53-targeting miRNAs (miR-25, miR-15a, miR-16, miR-92, miR-125b, miR-141, and miR-200a), while 2,3,7,8-tetrachlorodibenzo-rho-dioxin (TCDD) upregulated two of them (miR-25 and miR-92) in MM cells. Benzo(a)pyrene 50-64 microRNA 25 Homo sapiens 129-135 26550209-11 2015 The subgroup analysis showed that high expressed miR-25 could worsen OS in Chinese patients with pooled HR of 1.895 (95% CI 1.096-2.693, P=0.007). Osmium 69-71 microRNA 25 Homo sapiens 49-55 25764156-8 2015 MCU mRNA level was reversely correlated with miR-25 under the exposure of H2O2, and MCU protein level was largely decreased by miR-25 overexpression. Hydrogen Peroxide 74-78 microRNA 25 Homo sapiens 45-51 25764156-10 2015 MiR-25 significantly decreased H2O2-induced elevation of mitochondrial Ca2+ concentration, which is likely to be the result of decreased activity of MCU. Hydrogen Peroxide 31-35 microRNA 25 Homo sapiens 0-6 25575057-3 2015 The potential proliferation-stimulating function of miR-25 was analyzed by MTT assay in HeLa cells. monooxyethylene trimethylolpropane tristearate 75-78 microRNA 25 Homo sapiens 52-58 25451224-0 2014 miR-25 alleviates polyQ-mediated cytotoxicity by silencing ATXN3. polyglutamine 18-23 microRNA 25 Homo sapiens 0-6 25576360-4 2015 In addition, BTG expression was found negatively correlated with miR-25a expression in the both tissues and cells. beta-2'-deoxythioguanosine 13-16 microRNA 25 Homo sapiens 65-71 25575057-6 2015 MTT assay showed that miR-25 over-expression promoted the proliferation of HeLa cells. monooxyethylene trimethylolpropane tristearate 0-3 microRNA 25 Homo sapiens 22-28 24670661-5 2014 Whereas adeno-associated virus 9 (AAV9)-mediated overexpression of miR-25 in vivo resulted in a significant loss of contractile function, injection of an antisense oligonucleotide (antagomiR) against miR-25 markedly halted established heart failure in a mouse model, improving cardiac function and survival relative to a control antagomiR oligonucleotide. Oligonucleotides 164-179 microRNA 25 Homo sapiens 200-206 25550809-3 2014 In SCLC cell line H510A cells, endogenous miR-25 was downregulated by stable transfection of antisense oligonucleotide of miR-25 (miR-25-as). Oligonucleotides 103-118 microRNA 25 Homo sapiens 42-48 25550809-3 2014 In SCLC cell line H510A cells, endogenous miR-25 was downregulated by stable transfection of antisense oligonucleotide of miR-25 (miR-25-as). Oligonucleotides 103-118 microRNA 25 Homo sapiens 122-128 25550809-3 2014 In SCLC cell line H510A cells, endogenous miR-25 was downregulated by stable transfection of antisense oligonucleotide of miR-25 (miR-25-as). Oligonucleotides 103-118 microRNA 25 Homo sapiens 122-128 25550809-8 2014 Down-regulation of miR-25 in H510A cells significantly reduced cancer cell growth, invasive capability and resistance to cisplatin. Cisplatin 121-130 microRNA 25 Homo sapiens 19-25 25094038-5 2014 After validation by real-time PCR, all three members of the miR-106b-25 cluster (miR-106b, miR-93, and miR-25) were found to be markedly down-regulated during EMT in response to TGF-beta1, whereas these miRNAs were up-regulated by Sal B treatment in a dose-dependent manner. salvianolic acid B 231-236 microRNA 25 Homo sapiens 103-109 25026296-0 2014 MicroRNA-25 regulates chemoresistance-associated autophagy in breast cancer cells, a process modulated by the natural autophagy inducer isoliquiritigenin. isoliquiritigenin 136-153 microRNA 25 Homo sapiens 0-11 25026296-3 2014 Here, we report a novel function of isoliquiritigenin (ISL) as a natural inhibitor of autophagy-related miR-25 in killing drug-resistant breast cancer cells. isoliquiritigenin 36-53 microRNA 25 Homo sapiens 104-110 25026296-3 2014 Here, we report a novel function of isoliquiritigenin (ISL) as a natural inhibitor of autophagy-related miR-25 in killing drug-resistant breast cancer cells. isoliquiritigenin 55-58 microRNA 25 Homo sapiens 104-110 34396430-6 2021 The effects of miR-25 on NPC proliferation and apoptosis were evaluated using Cell Counting Kit-8 assay, 5-ethynyl-2"-deoxyuridine incorporation assay, and flow cytometry. 5-ethynyl-2'-deoxyuridine 105-130 microRNA 25 Homo sapiens 15-21 24270410-3 2014 Although all three miRs in the cluster contribute to the generation of doxorubicin resistance, miR-25 is the major contributor to this phenotype. Doxorubicin 71-82 microRNA 25 Homo sapiens 95-101 21953056-4 2012 In cultured cells, treatment with the Smoothened inhibitor, cyclopamine, reduced miR-25 expression, suggesting Hedgehog signaling stimulates miR-25 production. cyclopamine 60-71 microRNA 25 Homo sapiens 81-87 21953056-4 2012 In cultured cells, treatment with the Smoothened inhibitor, cyclopamine, reduced miR-25 expression, suggesting Hedgehog signaling stimulates miR-25 production. cyclopamine 60-71 microRNA 25 Homo sapiens 141-147 34863979-3 2022 DTX treatment promoted Ca2+-controlled autophagy and SIDT2 expression, resulting in lysosomal degradation of miR-25 in U937 cells. Docetaxel 0-3 microRNA 25 Homo sapiens 109-115