PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
33703997-7	2022	However, knockdown of AdipoR1 expression by AdipoR1-siRNA abolished leucine-induced upregulation of protein expressions of slow MyHC, AdipoR1, p-AMPK, PGC-1alpha and NRF1, mRNA expressions of MyHC I, MyHC IIa, AdipoR1, AMPKalpha2 and PGC-1alpha, ATP5G, TFAM and NRF1, and mtDNA level, as well as downregulation of protein expression of fast MyHC and mRNA expression of MyHC IIb.	Leucine	68-75	ATP synthase membrane subunit c locus 1	Homo sapiens	246-251
9247091-0	1997	Late-infantile ceroid-lipofuscinosis: lysine methylation of mitochondrial ATP synthase subunit c from lysosomal storage bodies.	Lysine	38-44	ATP synthase membrane subunit c locus 1	Homo sapiens	60-96
9247091-11	1997	Thus, it appears that specific methylation of lysine residue 43 of mitochondrial ATP synthase subunit c is probably a normal post-translational modification, and that the lysosomal storage of this protein in late-infantile, as well as in juvenile ceroid-lipofuscinosis, does not result from a defect in its methylation.	Lysine	46-52	ATP synthase membrane subunit c locus 1	Homo sapiens	67-103
7575423-0	1995	Mitochondrial ATP synthase subunit c stored in hereditary ceroid-lipofuscinosis contains trimethyl-lysine.	trimethyllysine	89-105	ATP synthase membrane subunit c locus 1	Homo sapiens	0-36
24558171-7	2014	Glycometabolism pathways network which was constructed by 4 glycometabolism pathways showed that adenosine triphosphate (ATP) synthase, H+transporting, mitochondrial F1 complex ATP5B, ATP5C1, ATP5D, and ATP5G1 had high degree related to ATP metabolism.	Adenosine Triphosphate	121-124	ATP synthase membrane subunit c locus 1	Homo sapiens	203-209
33412331-5	2021	Among these proteins, we identified 24 tyrosine phosphorylation sites in 17 mitochondrial proteins (AKAP1, VDAC1, VDAC2, VDAC3, LonP1, Hsp90, SLP2, PHB2, MIC60, UBA1, EF-Tu, LRPPRC, ACO2, OAT, ACAT1, ETFbeta and ATP5beta) as potential substrates for intramitochondrial Src using in silico prediction of tyrosine phospho-sites.	Tyrosine	39-47	ATP synthase membrane subunit c locus 1	Homo sapiens	212-220
24558171-7	2014	Glycometabolism pathways network which was constructed by 4 glycometabolism pathways showed that adenosine triphosphate (ATP) synthase, H+transporting, mitochondrial F1 complex ATP5B, ATP5C1, ATP5D, and ATP5G1 had high degree related to ATP metabolism.	Adenosine	97-106	ATP synthase membrane subunit c locus 1	Homo sapiens	203-209
31779731-5	2020	Furthermore, 1 0 % Arg supplementation significantly up-regulated PPAR-gamma coactivator-1alpha (PGC-1alpha), sirtuin 1 and cytochrome c (Cytc) protein expressions, increased PGC-1alpha, nuclear respiratory factor 1 (NRF1), mitochondria transcription factor B1 (TFB1M), Cytc and ATP synthase subunit C1 (ATP5G) mRNA levels and increased mitochondrial DNA content.	Arginine	19-22	ATP synthase membrane subunit c locus 1	Homo sapiens	304-309
27596602-4	2016	Nuclear expression of only the ATP8 gene with the ATP5G1 mitochondrial targeting sequence appended restored viability on Krebs cycle substrates and ATP synthesis capabilities but, failed to restore ATP hydrolysis and was insensitive to various inhibitors of oxidative phosphorylation.	krebs	121-126	ATP synthase membrane subunit c locus 1	Homo sapiens	50-56
27596602-4	2016	Nuclear expression of only the ATP8 gene with the ATP5G1 mitochondrial targeting sequence appended restored viability on Krebs cycle substrates and ATP synthesis capabilities but, failed to restore ATP hydrolysis and was insensitive to various inhibitors of oxidative phosphorylation.	Adenosine Triphosphate	31-34	ATP synthase membrane subunit c locus 1	Homo sapiens	50-56
24558171-7	2014	Glycometabolism pathways network which was constructed by 4 glycometabolism pathways showed that adenosine triphosphate (ATP) synthase, H+transporting, mitochondrial F1 complex ATP5B, ATP5C1, ATP5D, and ATP5G1 had high degree related to ATP metabolism.	Adenosine Triphosphate	177-180	ATP synthase membrane subunit c locus 1	Homo sapiens	203-209
23180398-1	2013	The neuronal ceroid-lipofuscinosis (NCL) are a heterogeneous group of neurodegenerative diseases characterized by the lysosomal accumulation of ceroid and lipofuscin with mitochondrial ATP synthase subunit C in various tissues.	Ceroid	13-19	ATP synthase membrane subunit c locus 1	Homo sapiens	171-207
23180398-1	2013	The neuronal ceroid-lipofuscinosis (NCL) are a heterogeneous group of neurodegenerative diseases characterized by the lysosomal accumulation of ceroid and lipofuscin with mitochondrial ATP synthase subunit C in various tissues.	Lipofuscin	20-30	ATP synthase membrane subunit c locus 1	Homo sapiens	171-207
22773120-5	2012	Importantly, miR-338 modulation of local COXIV and ATP5G1 expression has a marked effect on axonal ROS levels, as well as axonal growth.	ros	99-102	ATP synthase membrane subunit c locus 1	Homo sapiens	51-57