PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
6601061-1	1983	Conjugates were prepared by carbodiimide-mediated coupling of adipic acid hydrazide derivatives of Haemophilus influenzae type b (Hib), Escherichia coli K100, and pneumococcal 6A (Pn6A) polysaccharides with tetanus toxoid (TT), as an example of a "useful" carrier, and horseshoe crab hemocyanin (HCH), as an example of a "nonsense" carrier.	Carbodiimides	28-40	C1q-like 3	Mus musculus	153-157
6601061-1	1983	Conjugates were prepared by carbodiimide-mediated coupling of adipic acid hydrazide derivatives of Haemophilus influenzae type b (Hib), Escherichia coli K100, and pneumococcal 6A (Pn6A) polysaccharides with tetanus toxoid (TT), as an example of a "useful" carrier, and horseshoe crab hemocyanin (HCH), as an example of a "nonsense" carrier.	6-hydrazinyl-6-oxohexanoic acid	62-83	C1q-like 3	Mus musculus	153-157
6601061-7	1983	The K100-HCH conjugate was more immunogenic than the K100-TT conjugate and elicited anti-Hib responses similar to the Hib conjugates after the third injection.	alpha-hexachlorocyclohexane	9-12	C1q-like 3	Mus musculus	4-8
31676569-4	2020	In this study, CTRP13 treatment improved endothelium-dependent relaxation in the aortae and mesenteric arteries of both db/db mice and STZ-injected mice.	Streptozocin	135-138	C1q-like 3	Mus musculus	15-21
31676569-5	2020	CTRP13 supplement also rescued the impaired endothelium-dependent relaxation ex vivo in the db/db mouse aortae and in high glucose (HG)-treated mouse aortae.	Glucose	123-130	C1q-like 3	Mus musculus	0-6
31676569-6	2020	Additionally, CTRP13 treatment reduced reactive oxygen species overproduction and improved nitric oxide (NO) production and eNOS coupling in the aortae of diabetic mice and in HG-treated HUVECs.	Oxygen	48-54	C1q-like 3	Mus musculus	14-20
31676569-6	2020	Additionally, CTRP13 treatment reduced reactive oxygen species overproduction and improved nitric oxide (NO) production and eNOS coupling in the aortae of diabetic mice and in HG-treated HUVECs.	Nitric Oxide	91-103	C1q-like 3	Mus musculus	14-20
30222079-1	2019	C1q/tumor necrosis factor-related protein 13 (CTRP13) is a secreted adipokine that can ameliorate abnormal glucose and lipid metabolism.	Glucose	107-114	C1q-like 3	Mus musculus	46-52
30228187-3	2018	We found that C1ql3 predominantly inhibits exendin-4- and cAMP-stimulated insulin secretion from mouse and human islets.	Cyclic AMP	58-62	C1q-like 3	Mus musculus	14-19
26089941-2	2015	A sesquiterpene lactone 1-beta,10-Epoxy-6-hydroxy-1,10H-inunolide (K100) was isolated through "bioassay-guided fractionation" from Cota palaestina subsp.	sesquiterpene lactone 1-beta,10-epoxy-6-hydroxy-1,10h-inunolide	2-65	C1q-like 3	Mus musculus	67-71
26089941-5	2015	Molecular docking in silico suggested that K100, having highly analogous structure as parthenolide (PTL), an anticancer compound, could bind PTL target proteins at similar positions and with comparable binding affinities.	parthenolide	86-98	C1q-like 3	Mus musculus	43-47
23638159-1	2013	C1q/TNF-related protein 13 (CTRP13), a hormone secreted by adipose tissue (adipokines), helps regulate glucose metabolism in peripheral tissues.	Glucose	103-110	C1q-like 3	Mus musculus	0-26
23638159-1	2013	C1q/TNF-related protein 13 (CTRP13), a hormone secreted by adipose tissue (adipokines), helps regulate glucose metabolism in peripheral tissues.	Glucose	103-110	C1q-like 3	Mus musculus	28-34
21378161-0	2011	Metabolic regulation by C1q/TNF-related protein-13 (CTRP13): activation OF AMP-activated protein kinase and suppression of fatty acid-induced JNK signaling.	Fatty Acids	123-133	C1q-like 3	Mus musculus	24-50
21378161-0	2011	Metabolic regulation by C1q/TNF-related protein-13 (CTRP13): activation OF AMP-activated protein kinase and suppression of fatty acid-induced JNK signaling.	Fatty Acids	123-133	C1q-like 3	Mus musculus	52-58
21378161-7	2011	The insulin-sensitizing drug rosiglitazone also increases the expression of CTRP13 in adipocytes, which correlates with the insulin-sensitizing action of CTRP13.	Rosiglitazone	29-42	C1q-like 3	Mus musculus	76-82
21378161-7	2011	The insulin-sensitizing drug rosiglitazone also increases the expression of CTRP13 in adipocytes, which correlates with the insulin-sensitizing action of CTRP13.	Rosiglitazone	29-42	C1q-like 3	Mus musculus	154-160
21378161-8	2011	In a heterologous expression system, CTRP13 is secreted as a disulfide-linked oligomeric protein.	Disulfides	61-70	C1q-like 3	Mus musculus	37-43
21378161-11	2011	Functional studies using purified recombinant protein demonstrated that CTRP13 is an adipokine that promotes glucose uptake in adipocytes, myotubes, and hepatocytes via activation of the AMPK signaling pathway.	Glucose	109-116	C1q-like 3	Mus musculus	72-78
21378161-13	2011	Further, CTRP13 reduces glucose output in hepatocytes by inhibiting the mRNA expression of gluconeogenic enzymes, glucose-6-phosphatase and the cytosolic form of phosphoenolpyruvate carboxykinase.	Glucose	24-31	C1q-like 3	Mus musculus	9-15