PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9500792-0	1998	Acidic sphingomyelinase (ASM) is necessary for fas-induced GD3 ganglioside accumulation and efficient apoptosis of lymphoid cells.	ammonium ferrous sulfate	47-50	sphingomyelin phosphodiesterase 1	Homo sapiens	25-28	
9500792-0	1998	Acidic sphingomyelinase (ASM) is necessary for fas-induced GD3 ganglioside accumulation and efficient apoptosis of lymphoid cells.	ammonium ferrous sulfate	47-50	GRDX	Homo sapiens	59-62	
9500792-0	1998	Acidic sphingomyelinase (ASM) is necessary for fas-induced GD3 ganglioside accumulation and efficient apoptosis of lymphoid cells.	Gangliosides	63-74	sphingomyelin phosphodiesterase 1	Homo sapiens	25-28	
9500792-0	1998	Acidic sphingomyelinase (ASM) is necessary for fas-induced GD3 ganglioside accumulation and efficient apoptosis of lymphoid cells.	Gangliosides	63-74	GRDX	Homo sapiens	59-62	
9500792-1	1998	Ceramides deriving from sphingomyelin hydrolysis are important mediators of apoptotic signals originating from Fas (APO-1/CD95).	Ceramides	0-9	Fas cell surface death receptor	Homo sapiens	116-121	
9500792-1	1998	Ceramides deriving from sphingomyelin hydrolysis are important mediators of apoptotic signals originating from Fas (APO-1/CD95).	Ceramides	0-9	Fas cell surface death receptor	Homo sapiens	122-126	
9500792-1	1998	Ceramides deriving from sphingomyelin hydrolysis are important mediators of apoptotic signals originating from Fas (APO-1/CD95).	Sphingomyelins	24-37	Fas cell surface death receptor	Homo sapiens	116-121	
9500792-1	1998	Ceramides deriving from sphingomyelin hydrolysis are important mediators of apoptotic signals originating from Fas (APO-1/CD95).	Sphingomyelins	24-37	Fas cell surface death receptor	Homo sapiens	122-126	
9500792-5	1998	NPD lymphoblasts also fail to accumulate GD3 ganglioside, a downstream mediator of ceramide-induced cell death (De Maria, R., L. Lenti, F. Malisan, F. D"Agostino, B. Tomassini, A. Zeuner, M.R.	Ceramides	83-91	GRDX	Homo sapiens	41-44	
9500792-10	1998	Inefficient apoptosis is due to lack of ASM activity, because proximal signaling from Fas in NPD lymphoblasts is not impaired and apoptosis can be efficiently triggered by passing the ASM defect with exogenous ceramides.	Ceramides	210-219	sphingomyelin phosphodiesterase 1	Homo sapiens	184-187