PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9244169-3	1996	Further studies were performed to determine whether human chorionic gonadotropin (hCG) can interact with the insulin-like growth factor-I (IGF-I) signaling pathway to increase tyrosine phosphorylation of IRS-I.	Tyrosine	176-184	hypertrichosis 2 (generalised, congenital)	Homo sapiens	82-85	
9244169-7	1996	The increased tyrosine phosphorylation of IRS-I seen in response to IGF-I stimulation following treatment with either hCG or forskolin was not due to an increase in IRS-I content.	Tyrosine	14-22	hypertrichosis 2 (generalised, congenital)	Homo sapiens	118-121	
9244169-9	1996	Thus, we conclude that hCG/LH and IGF-I signaling pathways "cross-talk" to increase the levels of IRS-I tyrosine phosphorylation.	Tyrosine	104-112	hypertrichosis 2 (generalised, congenital)	Homo sapiens	23-26