PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8982732-0	1996	Flupirtine increases the levels of glutathione and Bc1-2 in hNT (human Ntera/D1) neurons: mode of action of the drug-mediated anti-apoptotic effect.	flupirtine	0-10	charged multivesicular body protein 2A	Homo sapiens	51-56	
8982732-5	1996	During Glu/NMDA-mediated apoptosis the levels of the intracellular anti-apoptotic agents Bc1-2 and glutathione dropped by more than 50%.	Glutamic Acid	7-10	charged multivesicular body protein 2A	Homo sapiens	89-94	
8982732-5	1996	During Glu/NMDA-mediated apoptosis the levels of the intracellular anti-apoptotic agents Bc1-2 and glutathione dropped by more than 50%.	N-Methylaspartate	11-15	charged multivesicular body protein 2A	Homo sapiens	89-94	
8982732-6	1996	Flupirtine completely abolished this reduction of Bc1-2 and glutathione level at a concentration of 10 microM.	flupirtine	0-10	charged multivesicular body protein 2A	Homo sapiens	50-55	
8982732-7	1996	In the presence of 3 microM flupirtine a > 6-fold increase of the Bc1-2 (B-cell leukemia/lymphoma-2) level was observed in hNT neurons.	flupirtine a	28-40	charged multivesicular body protein 2A	Homo sapiens	69-74	
8982732-9	1996	We conclude that the Glu/NMDA-mediated neuronal cell death in vitro is controlled at least partially by Bc1-2 and glutathione.	Glutamic Acid	21-24	charged multivesicular body protein 2A	Homo sapiens	104-109	
8982732-9	1996	We conclude that the Glu/NMDA-mediated neuronal cell death in vitro is controlled at least partially by Bc1-2 and glutathione.	N-Methylaspartate	25-29	charged multivesicular body protein 2A	Homo sapiens	104-109