PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8819070-2	1996	The t(15;17) leads to the formation of two reciprocal fusion genes, PML/RARA on chromosome 15 and RARA/PML on chromosome 17; it is responsible for the unique response of the disease to retinoic acid (ATRA) treatment.	Tretinoin	185-198	PML nuclear body scaffold	Homo sapiens	68-71	
8819070-2	1996	The t(15;17) leads to the formation of two reciprocal fusion genes, PML/RARA on chromosome 15 and RARA/PML on chromosome 17; it is responsible for the unique response of the disease to retinoic acid (ATRA) treatment.	Tretinoin	185-198	retinoic acid receptor alpha	Homo sapiens	72-76	
8819070-2	1996	The t(15;17) leads to the formation of two reciprocal fusion genes, PML/RARA on chromosome 15 and RARA/PML on chromosome 17; it is responsible for the unique response of the disease to retinoic acid (ATRA) treatment.	Tretinoin	185-198	retinoic acid receptor alpha	Homo sapiens	98-102	
8819070-2	1996	The t(15;17) leads to the formation of two reciprocal fusion genes, PML/RARA on chromosome 15 and RARA/PML on chromosome 17; it is responsible for the unique response of the disease to retinoic acid (ATRA) treatment.	Tretinoin	185-198	PML nuclear body scaffold	Homo sapiens	103-106	
8819070-2	1996	The t(15;17) leads to the formation of two reciprocal fusion genes, PML/RARA on chromosome 15 and RARA/PML on chromosome 17; it is responsible for the unique response of the disease to retinoic acid (ATRA) treatment.	Tretinoin	200-204	PML nuclear body scaffold	Homo sapiens	68-71	
8819070-2	1996	The t(15;17) leads to the formation of two reciprocal fusion genes, PML/RARA on chromosome 15 and RARA/PML on chromosome 17; it is responsible for the unique response of the disease to retinoic acid (ATRA) treatment.	Tretinoin	200-204	retinoic acid receptor alpha	Homo sapiens	72-76	
8819070-2	1996	The t(15;17) leads to the formation of two reciprocal fusion genes, PML/RARA on chromosome 15 and RARA/PML on chromosome 17; it is responsible for the unique response of the disease to retinoic acid (ATRA) treatment.	Tretinoin	200-204	retinoic acid receptor alpha	Homo sapiens	98-102	
8819070-2	1996	The t(15;17) leads to the formation of two reciprocal fusion genes, PML/RARA on chromosome 15 and RARA/PML on chromosome 17; it is responsible for the unique response of the disease to retinoic acid (ATRA) treatment.	Tretinoin	200-204	PML nuclear body scaffold	Homo sapiens	103-106