PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8580427-2	1995	This phenotype is clearly different from classical paramyotonia congenita Eulenburg, which has been shown to be a sodium channelopathy resulting from mutations in the gene for the alpha-subunit of the human skeletal muscle sodium channel gene (SCN4A).	Sodium	114-120	sodium voltage-gated channel alpha subunit 4	Homo sapiens	244-249	
8580427-2	1995	This phenotype is clearly different from classical paramyotonia congenita Eulenburg, which has been shown to be a sodium channelopathy resulting from mutations in the gene for the alpha-subunit of the human skeletal muscle sodium channel gene (SCN4A).	Sodium	223-229	sodium voltage-gated channel alpha subunit 4	Homo sapiens	244-249