PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7966593-2	1994	Treatment of cells with classical NF-kappa B inducers such as tumor necrosis factor, interleukin-1, phorbol myristate acetate, and lipopolysaccharide results in MAD3 degradation followed by nuclear translocation of NF-kappa B.	Tetradecanoylphorbol Acetate	100-125	nuclear factor kappa B subunit 1	Homo sapiens	34-44	
7966593-2	1994	Treatment of cells with classical NF-kappa B inducers such as tumor necrosis factor, interleukin-1, phorbol myristate acetate, and lipopolysaccharide results in MAD3 degradation followed by nuclear translocation of NF-kappa B.	Tetradecanoylphorbol Acetate	100-125	NFKB inhibitor alpha	Homo sapiens	161-165	
7966593-2	1994	Treatment of cells with classical NF-kappa B inducers such as tumor necrosis factor, interleukin-1, phorbol myristate acetate, and lipopolysaccharide results in MAD3 degradation followed by nuclear translocation of NF-kappa B.	Tetradecanoylphorbol Acetate	100-125	nuclear factor kappa B subunit 1	Homo sapiens	215-225	
7966593-8	1994	On the other hand, Tax induces no apparent degradation of MAD3, although experiments using cycloheximide indicate that it decreases the half-life of MAD3.	Cycloheximide	91-104	NFKB inhibitor alpha	Homo sapiens	149-153