PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7833480-1	1995	Studies of neutrophil nicotinamide adenine dinucleotide phosphate (NADPH) oxidase activation in a cell-free system showed that the low molecular-weight guanosine triphosphatase (GTPase) Rac was required, and that Rap1a may participate in activation of the catalytic complex.	Adenine	35-42	Rac family small GTPase 2	Homo sapiens	186-189	
7833480-3	1995	In the present studies, oligonucleotide-directed mutagenesis was used to introduce a series of mutations into human rap1a or rac2 in the mammalian expression vector pSR alpha neo.	Oligonucleotides	24-39	RAP1A, member of RAS oncogene family	Homo sapiens	116-121	
7833480-3	1995	In the present studies, oligonucleotide-directed mutagenesis was used to introduce a series of mutations into human rap1a or rac2 in the mammalian expression vector pSR alpha neo.	Oligonucleotides	24-39	Rac family small GTPase 2	Homo sapiens	125-129	
7833480-12	1995	production by the differentiated cell line expressing GTPase-defective V12 Rap1a was also significantly inhibited, a finding that is consistent with a requirement for cycling between guanosine diphosphate- and GTP-bound forms of Rap1a for continuous NADPH oxidase activation in intact neutrophils.	Guanosine Diphosphate	183-204	RAP1A, member of RAS oncogene family	Homo sapiens	75-80	
7833480-12	1995	production by the differentiated cell line expressing GTPase-defective V12 Rap1a was also significantly inhibited, a finding that is consistent with a requirement for cycling between guanosine diphosphate- and GTP-bound forms of Rap1a for continuous NADPH oxidase activation in intact neutrophils.	Guanosine Diphosphate	183-204	RAP1A, member of RAS oncogene family	Homo sapiens	229-234	
7833480-12	1995	production by the differentiated cell line expressing GTPase-defective V12 Rap1a was also significantly inhibited, a finding that is consistent with a requirement for cycling between guanosine diphosphate- and GTP-bound forms of Rap1a for continuous NADPH oxidase activation in intact neutrophils.	Guanosine Triphosphate	54-57	RAP1A, member of RAS oncogene family	Homo sapiens	75-80	
7833480-12	1995	production by the differentiated cell line expressing GTPase-defective V12 Rap1a was also significantly inhibited, a finding that is consistent with a requirement for cycling between guanosine diphosphate- and GTP-bound forms of Rap1a for continuous NADPH oxidase activation in intact neutrophils.	Guanosine Triphosphate	54-57	RAP1A, member of RAS oncogene family	Homo sapiens	229-234