PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
35431993-4	2022	The absence of extracellular Ca2+ influx after immersion into nominally zero Ca2+ solution, or the addition of nifedipine, significantly inhibited the contractile responses (p < 0.05 for all) after stimulation with carbachol (1 microM), histamine (100 microM), 5-hydroxytryptamine (100 microM), neurokinin-A (300 nM), prostaglandin E2 (10 microM), and angiotensin II (100 nM).	Nifedipine	111-121	tachykinin precursor 1	Homo sapiens	295-307	
35431993-4	2022	The absence of extracellular Ca2+ influx after immersion into nominally zero Ca2+ solution, or the addition of nifedipine, significantly inhibited the contractile responses (p < 0.05 for all) after stimulation with carbachol (1 microM), histamine (100 microM), 5-hydroxytryptamine (100 microM), neurokinin-A (300 nM), prostaglandin E2 (10 microM), and angiotensin II (100 nM).	Nifedipine	111-121	angiotensinogen	Homo sapiens	352-366	
35431993-4	2022	The absence of extracellular Ca2+ influx after immersion into nominally zero Ca2+ solution, or the addition of nifedipine, significantly inhibited the contractile responses (p < 0.05 for all) after stimulation with carbachol (1 microM), histamine (100 microM), 5-hydroxytryptamine (100 microM), neurokinin-A (300 nM), prostaglandin E2 (10 microM), and angiotensin II (100 nM).	Carbachol	215-224	tachykinin precursor 1	Homo sapiens	295-307	
35431993-4	2022	The absence of extracellular Ca2+ influx after immersion into nominally zero Ca2+ solution, or the addition of nifedipine, significantly inhibited the contractile responses (p < 0.05 for all) after stimulation with carbachol (1 microM), histamine (100 microM), 5-hydroxytryptamine (100 microM), neurokinin-A (300 nM), prostaglandin E2 (10 microM), and angiotensin II (100 nM).	Carbachol	215-224	angiotensinogen	Homo sapiens	352-366