PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
35306537-4	2022	Levels of phosphorylated IkBalpha and p65 significantly rose in mdx/mTR-/- dystrophic hearts and Wt1 expression declined in the epicardium of mdx/mTR-/- mice when nuclear factor kappaB (NF-kappaB) and inflammation were inhibited by metformin.	Metformin	232-241	nuclear factor of kappa light polypeptide gene enhancer in B cells inhibitor, alpha	Mus musculus	25-33	
35306537-4	2022	Levels of phosphorylated IkBalpha and p65 significantly rose in mdx/mTR-/- dystrophic hearts and Wt1 expression declined in the epicardium of mdx/mTR-/- mice when nuclear factor kappaB (NF-kappaB) and inflammation were inhibited by metformin.	Metformin	232-241	v-rel reticuloendotheliosis viral oncogene homolog A (avian)	Mus musculus	38-41	
35306537-4	2022	Levels of phosphorylated IkBalpha and p65 significantly rose in mdx/mTR-/- dystrophic hearts and Wt1 expression declined in the epicardium of mdx/mTR-/- mice when nuclear factor kappaB (NF-kappaB) and inflammation were inhibited by metformin.	Metformin	232-241	WT1 transcription factor	Mus musculus	97-100	
35306537-7	2022	Our study demonstrates that upregulation of Wt1 in epicardial cells contributes to fibrosis in dystrophic hearts and metformin-mediated inhibition of NF-kappaB can ameliorate the pathology, and thus showing clinical potential for dystrophic cardiomyopathy.	Metformin	117-126	WT1 transcription factor	Mus musculus	44-47	
35306537-7	2022	Our study demonstrates that upregulation of Wt1 in epicardial cells contributes to fibrosis in dystrophic hearts and metformin-mediated inhibition of NF-kappaB can ameliorate the pathology, and thus showing clinical potential for dystrophic cardiomyopathy.	Metformin	117-126	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	150-159