PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34894475-4	2021	Gclc deficiency compromised the activation of mammalian target of rapamycin-1 (mTOR) and expression of c-Myc transcription factors, abrogating the energy utilization and the metabolic reprogramming that allows beta-glucan-trained macrophages to switch to glycolysis and glutaminolysis.	beta-Glucans	210-221	MYC proto-oncogene, bHLH transcription factor	Homo sapiens	103-108