PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34890285-0	2022	Synergistic Inhibitory Effects of 5-Aza-2"-Deoxycytidine and Cisplatin on Urothelial Carcinoma Growth via Suppressing TGFBI-MAPK Signaling Pathways.	Cisplatin	61-70	transforming growth factor beta induced	Homo sapiens	118-123	
34890285-4	2022	The results showed that several key regulatory genes, such as interleukin 24(IL24), fibroblast growth factor 1(FGF1), and transforming growth factor beta-induced (TGFBI), were identified and may play critical roles in the synergy of DAC and CDDP in UC.	Cisplatin	241-245	transforming growth factor beta induced	Homo sapiens	122-161	
34890285-4	2022	The results showed that several key regulatory genes, such as interleukin 24(IL24), fibroblast growth factor 1(FGF1), and transforming growth factor beta-induced (TGFBI), were identified and may play critical roles in the synergy of DAC and CDDP in UC.	Cisplatin	241-245	transforming growth factor beta induced	Homo sapiens	163-168	
34890285-6	2022	Our results suggested that TGF-beta1 stimulates the phosphorylation levels of ERK1/2 and p38 via increasing TGFBI expression, TGFBI-MAPK signaling pathway plays an important role in the synergy of DAC and CDDP against UC.	Cisplatin	205-209	transforming growth factor beta induced	Homo sapiens	108-113	
34890285-6	2022	Our results suggested that TGF-beta1 stimulates the phosphorylation levels of ERK1/2 and p38 via increasing TGFBI expression, TGFBI-MAPK signaling pathway plays an important role in the synergy of DAC and CDDP against UC.	Cisplatin	205-209	transforming growth factor beta induced	Homo sapiens	126-131	
34890285-7	2022	Therefore, we revealed the synergistic mechanism of DAC and CDDP in UC, several key regulatory genes play critical roles in the synergy of combined treatment, and TGFBI-MAPK signaling pathway may be an important potential target of these two agents.	Cisplatin	60-64	transforming growth factor beta induced	Homo sapiens	163-168