PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34781065-7	2022	Here, we found that AHR and IDO1 expression levels were lower in endometriosis PBMCs; however, TCDD treatment increased AHR, FOXP3, IDO1, IL-6, and Treg levels in the endometriosis group (P <= 0.05-0.0001).	Polychlorinated Dibenzodioxins	95-99	aryl hydrocarbon receptor	Homo sapiens	20-23	
34781065-7	2022	Here, we found that AHR and IDO1 expression levels were lower in endometriosis PBMCs; however, TCDD treatment increased AHR, FOXP3, IDO1, IL-6, and Treg levels in the endometriosis group (P <= 0.05-0.0001).	Polychlorinated Dibenzodioxins	95-99	aryl hydrocarbon receptor	Homo sapiens	120-123	
34781065-9	2022	In conclusion, these results shed light on the probable mechanisms, through which AHR activation by chemical toxicants can impact inflammatory immune mediators involved in the development of endometriosis; also, these data support the idea that TCDD could promote endometriosis progression.	Polychlorinated Dibenzodioxins	245-249	aryl hydrocarbon receptor	Homo sapiens	82-85