PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34735871-5	2021	Mechanistically, LIGHT interacted with lymphotoxin-beta receptor (LTbetaR) to attenuate AKT pathway leading to downregulation of glucose transporter-4 (GLUT4) expression, which resulted in glucose uptake inhibition.	Glucose	189-196	lymphotoxin beta receptor	Homo sapiens	66-73	
34735871-0	2021	LIGHT (TNFSF14) inhibits glucose uptake of adipocytes by downregulating GLUT4 expression via AKT signaling pathway.	Glucose	25-32	TNF superfamily member 14	Homo sapiens	7-14	
34735871-0	2021	LIGHT (TNFSF14) inhibits glucose uptake of adipocytes by downregulating GLUT4 expression via AKT signaling pathway.	Glucose	25-32	solute carrier family 2 member 4	Homo sapiens	72-77	
34735871-0	2021	LIGHT (TNFSF14) inhibits glucose uptake of adipocytes by downregulating GLUT4 expression via AKT signaling pathway.	Glucose	25-32	AKT serine/threonine kinase 1	Homo sapiens	93-96	
34735871-2	2021	In order to investigate the effects of Lymphotoxin-like inducible protein that competes with glycoprotein D for herpesvirus entry on T cells (LIGHT) on glucose metabolism, we performed the present study.	Glucose	152-159	atypical chemokine receptor 1 (Duffy blood group)	Homo sapiens	93-107	
34735871-5	2021	Mechanistically, LIGHT interacted with lymphotoxin-beta receptor (LTbetaR) to attenuate AKT pathway leading to downregulation of glucose transporter-4 (GLUT4) expression, which resulted in glucose uptake inhibition.	Glucose	129-136	lymphotoxin beta receptor	Homo sapiens	39-64	
34735871-5	2021	Mechanistically, LIGHT interacted with lymphotoxin-beta receptor (LTbetaR) to attenuate AKT pathway leading to downregulation of glucose transporter-4 (GLUT4) expression, which resulted in glucose uptake inhibition.	Glucose	129-136	lymphotoxin beta receptor	Homo sapiens	66-73	
34735871-5	2021	Mechanistically, LIGHT interacted with lymphotoxin-beta receptor (LTbetaR) to attenuate AKT pathway leading to downregulation of glucose transporter-4 (GLUT4) expression, which resulted in glucose uptake inhibition.	Glucose	129-136	AKT serine/threonine kinase 1	Homo sapiens	88-91	
34735871-5	2021	Mechanistically, LIGHT interacted with lymphotoxin-beta receptor (LTbetaR) to attenuate AKT pathway leading to downregulation of glucose transporter-4 (GLUT4) expression, which resulted in glucose uptake inhibition.	Glucose	129-136	solute carrier family 2 member 4	Homo sapiens	152-157	
34735871-5	2021	Mechanistically, LIGHT interacted with lymphotoxin-beta receptor (LTbetaR) to attenuate AKT pathway leading to downregulation of glucose transporter-4 (GLUT4) expression, which resulted in glucose uptake inhibition.	Glucose	189-196	lymphotoxin beta receptor	Homo sapiens	39-64	
34735871-5	2021	Mechanistically, LIGHT interacted with lymphotoxin-beta receptor (LTbetaR) to attenuate AKT pathway leading to downregulation of glucose transporter-4 (GLUT4) expression, which resulted in glucose uptake inhibition.	Glucose	189-196	AKT serine/threonine kinase 1	Homo sapiens	88-91	
34735871-5	2021	Mechanistically, LIGHT interacted with lymphotoxin-beta receptor (LTbetaR) to attenuate AKT pathway leading to downregulation of glucose transporter-4 (GLUT4) expression, which resulted in glucose uptake inhibition.	Glucose	189-196	solute carrier family 2 member 4	Homo sapiens	152-157	
34735871-6	2021	In summary, our findings revealed LIGHT-LTbetaR-AKT-GLUT4 axis as a regulator of glucose uptake in adipose tissue, which suggested the pivotal role of LIGHT in maintaining glucose homeostasis.	Glucose	81-88	AKT serine/threonine kinase 1	Homo sapiens	48-51	
34735871-6	2021	In summary, our findings revealed LIGHT-LTbetaR-AKT-GLUT4 axis as a regulator of glucose uptake in adipose tissue, which suggested the pivotal role of LIGHT in maintaining glucose homeostasis.	Glucose	81-88	solute carrier family 2 member 4	Homo sapiens	52-57