PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33228663-5	2020	In the in vitro cellular experiment, we transfected siRNAs, oligonucleotides or plasmids into ethanol-induced AML-12 mouse hepatocytes to alter NEAT1 and miR-129-5p expression, and inflammatory factors and lipid content were determined.	Ethanol	94-101	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	144-149	
33228663-5	2020	In the in vitro cellular experiment, we transfected siRNAs, oligonucleotides or plasmids into ethanol-induced AML-12 mouse hepatocytes to alter NEAT1 and miR-129-5p expression, and inflammatory factors and lipid content were determined.	Ethanol	94-101	microRNA 1295a	Homo sapiens	154-164	
33228663-10	2020	Inhibited NEAT1 or elevated miR-129-5p suppressed the elevated lipid metabolism and restrained inflammation reaction in ethanol-stimulated AML-12 cells.	Ethanol	120-127	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	10-15	
33228663-10	2020	Inhibited NEAT1 or elevated miR-129-5p suppressed the elevated lipid metabolism and restrained inflammation reaction in ethanol-stimulated AML-12 cells.	Ethanol	120-127	microRNA 1295a	Homo sapiens	28-38	
33228663-11	2020	The promoted miR-129-5p and inhibited NEAT1 could improve the liver function and repress blood lipid, inflammation reaction, hepatocyte apoptosis and liver fibrosis in ethanol-induced ASH mice.	Ethanol	168-175	microRNA 1295a	Homo sapiens	13-23	
33228663-11	2020	The promoted miR-129-5p and inhibited NEAT1 could improve the liver function and repress blood lipid, inflammation reaction, hepatocyte apoptosis and liver fibrosis in ethanol-induced ASH mice.	Ethanol	168-175	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	38-43