PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33103444-4	2020	In this study, we demonstrated that cisplatin activates AMPK (Thr172 phosphorylation) in cultured renal tubular epithelial cells in a time-dependent manner, which is associated with p53 phosphorylation.	Cisplatin	36-45	transformation related protein 53, pseudogene	Mus musculus	182-185	
33103444-5	2020	Compound C, a selective AMPK inhibitor, suppressed cisplatin-induced AMPK activation, p53 phosphorylation, Bax induction and caspase 3 activation.	Cisplatin	51-60	transformation related protein 53, pseudogene	Mus musculus	86-89	
33103444-6	2020	Furthermore, silencing AMPK expression by siRNA attenuated cisplatin-induced p53 phosphorylation, Bax induction, and caspase 3 activation.	Cisplatin	59-68	transformation related protein 53, pseudogene	Mus musculus	77-80	
33103444-7	2020	In a mouse model of cisplatin-induced kidney injury, compound C inhibited p53 phosphorylation, Bax expression, caspase 3 activation, and apoptosis, protecting the kidney from injury and dysfunction.	Cisplatin	20-29	transformation related protein 53, pseudogene	Mus musculus	74-77	
33103444-8	2020	Taken together, these results suggest that AMPK-p53-Bax signaling pathway plays a crucial role in cisplatin-induced tubular epithelial cell apoptosis.	Cisplatin	98-107	transformation related protein 53, pseudogene	Mus musculus	48-51