PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32890923-6	2020	Cd exposure inhibited phagocytic activity of chicken peritoneal macrophages, and promoted transcriptional status of pro-inflammatory cytokines (IL-1beta, IL-6 and TNF-alpha) in both unactivated macrophages and cells in response to lipopolysaccharide (LPS) stimuli.	Cadmium	0-2	interleukin 1, beta	Gallus gallus	144-152	
32890923-6	2020	Cd exposure inhibited phagocytic activity of chicken peritoneal macrophages, and promoted transcriptional status of pro-inflammatory cytokines (IL-1beta, IL-6 and TNF-alpha) in both unactivated macrophages and cells in response to lipopolysaccharide (LPS) stimuli.	Cadmium	0-2	interleukin 6	Gallus gallus	154-158	
32890923-6	2020	Cd exposure inhibited phagocytic activity of chicken peritoneal macrophages, and promoted transcriptional status of pro-inflammatory cytokines (IL-1beta, IL-6 and TNF-alpha) in both unactivated macrophages and cells in response to lipopolysaccharide (LPS) stimuli.	Cadmium	0-2	lipopolysaccharide induced TNF factor	Gallus gallus	163-172	
32890923-10	2020	In addition, NAC pretreatment promoted the phagocytic activity of Cd-exposed chicken peritoneal macrophages, and significantly inhibited expression of pro-inflammatory factors (IL-1beta, IL-6 and TNF-alpha) in both Cd-exposed macrophages and Cd-treated cells in response to LPS stimuli.	Acetylcysteine	13-16	interleukin 1, beta	Gallus gallus	177-185	
32890923-10	2020	In addition, NAC pretreatment promoted the phagocytic activity of Cd-exposed chicken peritoneal macrophages, and significantly inhibited expression of pro-inflammatory factors (IL-1beta, IL-6 and TNF-alpha) in both Cd-exposed macrophages and Cd-treated cells in response to LPS stimuli.	Acetylcysteine	13-16	interleukin 6	Gallus gallus	187-191	
32890923-10	2020	In addition, NAC pretreatment promoted the phagocytic activity of Cd-exposed chicken peritoneal macrophages, and significantly inhibited expression of pro-inflammatory factors (IL-1beta, IL-6 and TNF-alpha) in both Cd-exposed macrophages and Cd-treated cells in response to LPS stimuli.	Acetylcysteine	13-16	lipopolysaccharide induced TNF factor	Gallus gallus	196-205	
32890923-10	2020	In addition, NAC pretreatment promoted the phagocytic activity of Cd-exposed chicken peritoneal macrophages, and significantly inhibited expression of pro-inflammatory factors (IL-1beta, IL-6 and TNF-alpha) in both Cd-exposed macrophages and Cd-treated cells in response to LPS stimuli.	Cadmium	66-68	lipopolysaccharide induced TNF factor	Gallus gallus	196-205